• 제목/요약/키워드: nonregenerative

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A case of protein losing nephropathy in a dog infected with canine Babesia gibsoni

  • Kim, Ha-Jung;Park, Chul;Jung, Dong-In;Kang, Byeong-Teck;Kim, Ju-Won;Lim, Chae-Young;Park, Hee-Myung
    • 대한수의학회지
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    • 제46권1호
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    • pp.77-81
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    • 2006
  • A 1-year and 6-month-old, intact male, Shih-tzu dog was referred to the Konkuk University Veterinary Teaching Hospital, due to ascite, melena, severe anemia, and polyuria/polydipsia. Complete blood count showed moderate leukocytosis, lymphocytosis, monocytosis, and microcytic hypochromic nonregenerative anemia. On serum biochemistry profiles, hypoalbuminemia and hypoproteinemia were observed, and proteinuria was detected on urinalysis. Pleural effusion and mild ascites were noted in radiological findings, and bilateral nephromegaly was noted on ultrasonography. Babesia gibsoni was detected by PCR. Babesiosis and glomerular disease was treated at a time with prednisolone and buparvaquone. This therapeutic regime was very efficacious and clinical sings were rapidly improved. Proteinuria and severe anemia were disappeared following therapy.

초고속 광파이버 전송시스템에서 색분산에 의한 전송거리 제한에 관한 연구 (A Study on the Transmission Length Limitation by Chromatic Dispersion in High Speed FOT스s)

  • 정은숙;김재평;정진호;김영권
    • 한국전자파학회지:전자파기술
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    • 제4권3호
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    • pp.18-29
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    • 1993
  • 장거리 초고속 단일모드 광파이버 전송 시스템에서, 색분산은 복조된 파형의 찌그러짐을 생기게 하며, 그 결과 수신된 신호 간 간섭을 일으켜 전송 시스템의 성능을 저하시킨다. 본 논문에서는 강도변조-직접검파 시스 댐의 색분산제한을위상변조에서 진폭변조로의 전환잡음의 영향을고려함으로서 연구하였다. 파이버 색분산에 의한 위상-진폭으로의 전환잡음을 잡음전력 스펙트럼 밀도를 유도함으로서 분석하였으며 위상-진폭 전환잡음을 피하기 위한 시스댐 전력손실과 레이저 선폭에 대한 요구조건을 수치해석을 통하여 구하였다.

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개의 단순 간내성 간문맥전신단락증의 영상진단학 및 치료 1예 (Radiographic and Ultrasonographic Diagnosis of Single Intrahepatic Portosystemic Shunt in a Dog)

  • 전혜영;장동우
    • 한국임상수의학회지
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    • 제20권4호
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    • pp.508-515
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    • 2003
  • A 4-month-old 5.7 kg male Golden retriever with history of seizure, depression, lethargy and anorexia was referred to Veterinary Medical Teaching Hospital, Chungbuk National University. Hematologic examination revealed microcytosis and nonregenerative anemia. Serum chemical values showed increased serum ammonia (423 $\mu$mol/L), ALP (1101 U/L), r-GTP (13.9 U/L) and CPK (1454 U/L), and decreased total protein (4.9 g/dl) and BUN (1.6 mg/dl). Microhepatia was shown in survey abdominal radiographs. Color doppler ultrasonographic examination revealed dilated tortuous vein with turbulent flow within liver parenchyma. Intraoperative jejunoportography and intraoperative ultrasonography confirmed the location and size of single intrahepatic shunt vessel in the left medial liver lobe. Also, the anomalous vessel entering the caudal vena cava was identified beneath the diaphragm. The shunting vessel was ligated with using an Ameroid constrictor. General conditions, hematologic and serum chemical values resolved gradually after surgery. One month after surgery abdominal radiograph showed normal gastric axis and it was consistent whit the normal size liver. Normal echogenecity of liver and enlargement of portal vein were shown in ultrasonography. It is assumed that survey radiography and ultrasonography are useful for diagnosis of single intrahepatic shunt in a dog and especially jejunoportography vein portography and intraoperative ultrasonography are suitable for confirmation of the anatomic location and size of the shunting vessels.

Transient Non-Regenerative Anemia in a Dog with Granulomatous Meningoencephalitis Following Leflunomide Treatment

  • Ga-Hyun Lim;Ju-Hyun An;Su-Min Park;Jeong-Hwa Lee;Ye-In Oh;Kyoung-Won Seo;Hwa-Young Youn
    • 한국임상수의학회지
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    • 제41권3호
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    • pp.165-169
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    • 2024
  • A 10-year-old, spayed female Maltese dog was tentatively diagnosed with granulomatous meningoencephalitis (GME) on magnetic resonance imaging. The meningoencephalitis was classified as aseptic GME because cerebral fluid analysis did not reveal an infectious aetiology. Two months after leflunomide treatment (Arava; Sanofi; 4 mg/kg/day), the patient developed non-regenerative, macrocytic, and normochromic anemia. As the patient's anaemia began after the administration of leflunomide, and other differentials for anaemia had been ruled out, the leflunomide was determined to be the cause and this treatment ended. After 15 days, the anaemia resolved spontaneously. This is the first report of reversible aplastic anaemia following treatment with leflunomide in a canine patient with GME.

만성 신부전에서 속발한 metastatic calcinosis circumscripta의 진단과 치료 1례 (A case report of diagnosis and treatment of metastatic calcinosis circumscripta secondary to chronic renal failure)

  • 박성준;박혜연;전소분;최호정;정성목;송근호;조종기;이영원;신상태;김명철;김덕환
    • 대한수의학회지
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    • 제45권3호
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    • pp.445-450
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    • 2005
  • A 1.35 kg, three-year-old, female, Yorkshire terrier was referred to the veterinary medical teaching hospital of Chungnam National University with an alopecia, scale, polydipsia and polyuria. During hospitalization, the patient revealed swelling and pain of all four foot pads. The hematology indicated nonregenerative anemia. Blood serum chemistry revealed elevation of BUN, serum creatinine and phosphorus contents. Radiographs of feet revealed increased radiodensity in the soft tissue of the foot pad. In ultrasonographs of abdomen, kidneys showed diffusely echodense renal cortex with loss of the normal corticomedullary boundary. Fine needle aspiration of the swollen pad cysts contained a amorphous basophillic chalky, white and pasty material. The culture result was negative for bacteria. In conclusion, it was diagnosed as metastatic calcinosis circumscripta secondary to chronic renal failure. An oral charcoal absorbent and aluminum hydroxide were used to treat this condition. After six weeks treatment, hyperphosphatemia was corrected and metastatic calcinosis circumscriptawas not presented any more.

개의 다발성 간외성 간문맥전신단락증의 진단 2예 (Diagnosis of Multiple Extrahepatic Portosystemic Shunt in Two Dogs)

  • 김주형;한성영;전혜영;김태훈;강지훈;한태성;나기정;양만표;김근형;강상철;김재훈;장동우
    • 한국임상수의학회지
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    • 제24권2호
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    • pp.269-275
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    • 2007
  • Two dogs referred to Veterinary Medical Center, Chungbuk National University diagnosed as multiple extrahepatic portosystemic shunt were reported. The first dog was a 20-month-old, 8 kg, male Cocker spaniel with history of peritoneal effusion, diarrhea, anorexia and stunted growth. The second dog was a 3-year-old, 13.4 kg, male Jindo with a history of severe depression. Hematologic examination of first dog revealed mild microcytosis and nonregenerative anemia. All of 2 cases, serum chemical values showed increase of serum ammonia, ALP, r-GTP and glucose. In survey radiography, microhepatia was apparent. In the color Doppler ultrasonographic examination, the first dog revealed a dilated tortuous vein communicating with caudal vena cava was observed near the left kidney and the second dog revealed numerous shunting vessels ventral to L5 and L6. Transcolonic portal scintigraphy of the first dog confirmed the presence of portosystemic shunt. In intraoperative jejunoportography, the first dog showed single congenital extrahepatic portosystemic shunt and multiple acquired extrahepatic portosystemic shunts. The second dog showed multiple acquired extrahepatic portosystemic shunts. In these dogs, the presence of congenital and acquried portosystemic shunts and histopathologic findings were considered to represent a combination of multiple extrahepatic portosystemic shunts and noncirrhotic portal hypertension or portal vein hypoplasia.