• 제목/요약/키워드: etiology.

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신생아 경련의 예후 (Outcomes of Neonatal Seizures)

  • 성인경
    • Neonatal Medicine
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    • 제16권1호
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    • pp.18-24
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    • 2009
  • Seizures are the most common clinical symptom of a neurologic insult and have long been recognized as an obvious marker of brain dysfunction in newborns. Presence of seizures in newborn infants may signify substantial risk for subsequent neurodevelopmental impairment including postneonatal epilepsy and death. The outcomes of seizures in neonates are determined mainly by the etiology of the seizures. Despite the decreasing trend of mortality of neonatal seizures, the prevalence of long-term neurodevelopmental sequelae in survivors has remained unchanged over time. Clinical studies have contributed to identifying significant prognostic factors for neurodevelopmental outcome. The underlying etiology of the seizures and electroencepaphalography background pattern are considered as most reliable early predictors of later neurologic sequelae. However, clinicians managing neonatal seizures are still challenged by difficult therapeutic and prognostic questions because of many unresolved issues in seizure recognition, terminology, relationships to the underlying brain lesion, effect of current management, particularly antiepileptic drugs on long-term outcomes. This review presents the prognosis of neonatal seizures, especially about mortality and neurodevelopmental deficit, and predictors of outcomes.

A case of Kikuchi-Fujimoto disease with autoimmune thyroiditis

  • Go, Eun Ji;Jung, You Jin;Han, Seung Beom;Suh, Byung Kyu;Kang, Jin Han
    • Clinical and Experimental Pediatrics
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    • 제55권11호
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    • pp.445-448
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    • 2012
  • Kikuchi-Fujimoto disease (KFD) is a benign self-limiting disease characterized by fever and lymphadenitis. The etiology and pathogenesis of KFD is unclear. However, two hypotheses have been suggested: a viral infection hypothesis and an autoimmune hypothesis. Several KFD patients with various types of autoimmune diseases have been reported, and these reports support the hypothesis for autoimmune pathogenesis of KFD. Here, we report the case of a 17-year-old female patient diagnosed with KFD and autoimmune thyroiditis. This case serves as additional evidence that the etiology of KFD is autoimmune origin.

Posttraumatic Giant Extradural Intradiploic Epidermoid Cysts of Posterior Cranial Fossa: Case Report and Review of the Literature

  • Enchev, Yavor;Kamenov, Bogidar;William, Alla;Karakostov, Vasil
    • Journal of Korean Neurosurgical Society
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    • 제49권1호
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    • pp.53-57
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    • 2011
  • We reported a unique case of posttraumatic giant infratentorial extradural intradiploic epidermoid cyst. A 54-year-old male, with a previous history of an open scalp injury and underlying linear skull fracture in the left occipital region in childhood, presented with a painful subcutaneous swelling, which had been developed gradually in the same region and moderate headache, nausea, vomiting and cerebellar ataxia. The duration of symptoms on admission was 3 months. Imaging studies revealed occipital bone destruction and giant extradural intradiploic lesion. The preoperative diagnosis was giant infratentorial extradural intradiploic epidermoid cyst. Surgery achieved total removal of the lesion, which was histologically confirmed and the postoperative course was uneventful. To our knowledge, this is the first case of giant infratentorial extradural intradiploic epidermoid cyst with a traumatic etiology described in the literature.

Ulnar Nerve Compression at Guyon's Canal by an Arteriovenous Malformation

  • Kim, Sung-Soo;Kim, Jae-Hoon;Kang, Hee-In;Lee, Seung-Jin
    • Journal of Korean Neurosurgical Society
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    • 제45권1호
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    • pp.57-59
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    • 2009
  • Guyon's canal at the wrist is not the common site of ulnar nerve compression. Ganglion, lipoma, anomalous tendon and muscles, trauma related to an occupation, arthritis, and carpal bone fracture can cause ulnar nerve compression at the wrist. However, ulnar nerve compression at Guyon's canal by vascular lesion is rare. Ulnar artery aneurysm, tortous ulnar artery, hemangioma, and thrombosis have been reported in the literature as vascular lesions. The authors experienced a case of ulnar nerve compression at Guyon's canal by an arteriovenous malformation (AVM) and the patient's symptom was improved after surgical resection. We can not easily predict vascular lesion as a cause of ulnar nerve compression at Guyon's canal. However, if there is not obvious etiology, we should consider vascular lesion as another possible etiology.

Unusual intraosseous transmigration of impacted tooth

  • Kumar, Santosh;Urala, Arun Srinivas;Kamath, Abhay Taranath;Jayaswal, Priyanka;Valiathan, Ashima
    • Imaging Science in Dentistry
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    • 제42권1호
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    • pp.47-54
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    • 2012
  • Transmigration of an impacted tooth through the symphyseal suture is a rare and special developmental anomaly of unknown etiology that is unique to the mandibular canine. Maxillary canine transmigration is even rarer. Transmigrated canines are particularly significant due to the aesthetic and functional importance. A maxillary lateral incisor crossing the mid-palatal suture has never been reported in the literature. The aim of this report is to present the first case of simultaneous transmigration of a lateral incisor and canine in the maxilla. The paper also reports four unusual cases of unilateral canine transmigration in the maxilla and mandible and successful eruption of one of the transmigrated mandibular canines following orthodontic traction. Etiology of transmigration and its clinical considerations are also discussed.

가와사끼병의 심혈관계 합병증 및 치료 (Cardiovascular complications after Kawasaki disease and its management)

  • 장기영
    • Clinical and Experimental Pediatrics
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    • 제51권5호
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    • pp.462-467
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    • 2008
  • Kawasaki disease is a systemic vasculitis of unknown etiology, usually occurring in infants and young children. Although the etiology of Kawasaki disease remains uncertain, its serious complicationssuch as giant aneurysm formation, coronary arterial stenotic lesions, and thrombotic occlusionhave been proven to cause myocardial ischemia or infarction in patients with Kawasaki disease. To prevent and treat these complications, several modes of therapyincluding long-term anticoagulation, interventional catheterization, and surgical treatmenthave been gradually developed. In this article, we review the cardiovascular complications following Kawasaki disease and the management thereof, which includes thrombolytic therapy, catheter intervention, and coronary artery bypass graft.

신생아의 급성신손상 (Acute Kidney Injury in the Newborn: Etiology, Pathophysiology and Diagnosis)

  • 김소영
    • Neonatal Medicine
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    • 제17권2호
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    • pp.161-167
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    • 2010
  • Acute kidney injury (AKI), formerly referred to as acute renal failure (ARF) is defined as the sudden impairment of kidney function (estimated from the glomerular filtration rate [GFR]) that results in the lack of excretion of waste products. More than 30 definitions of AKI exist in the literature, most of which are based on serum creatinine. Lack of a uniform and multidimensional AKI definition has led to failure to recognize significant renal injury, delays in treatment, and inability to generalize single-study results. The RIFLE criteria were developed to standardize the diagnosis of ARF and in the process the term AKI has been proposed to encompass the entire spectrum of the syndrome from minor changes in renal function to requirement for renal replacement therapy. Large prospective studies are needed to test definitions and to better understand risk factors, incidence, independent outcomes, and mechanisms that lead to poor short- and long-term outcomes. Early biomarkers of AKI need to be explored in critically ill neonates.

Meniere's Syndrome에서 나타나는 현훈증상의 침구치료 (The bibliographical study on the cause and etiology of vertigo(眩暈))

  • 정연탁;이병렬
    • 혜화의학회지
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    • 제11권1호
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    • pp.163-177
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    • 2002
  • This study has been carried out to investigate the cause and etiology of vertigo by referring to 47 literatures. The results were as follows ; 1. Vertigo is classified in Oriental Medicine into Exess Symptom-Complex(實證) and Deficiency Symptom-Complex(虛證). Exess Symptom-Complex(實證) is caused by flare-up the fire of the liver(肝陽上亢), phlegm-heat(痰火) and exogenous pathogenic factors(外邪). Deficiency Symptom-Complex(虛證) is caused by insufficiency of the yin of the kidney(腎陰不足) and deficiency of qi and blood(氣血虛). 2. The principles of vertigo treatments are pyongganjamyang(平肝潛陽), sohwasigpung(消火息風), boiggihyul(補益氣血), geonunbiui(建運脾胃), boigsinjeong(補益腎精), chungyangnoisu(充養腦髓), joseubgeodam(燥濕祛痰), geonblhwaui(建脾和胃). 3. Various Needling Treatment Methods of vertigo in the recent Oriental Medicine are presented such as Filiform Needle(鍼刺療法), Auricular Acupuncture Therapy(耳鍼療法), Scalp Acupuncture Therapy(頭鍼療法), Cataneous Needle Therapy(皮膚鍼療法), Aqua Acupuncture Therapy(水鍼療法), etc.

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구창(口瘡)의 병인병기(病因病機)와 침구치료(鍼灸治療)에 관(關)한 문헌적(文獻的) 고찰(考察) (A Study of Literature Review on the Etiology & Pathologic Mechanism and on the Acupuncture & Moxibustion Treatment for GU-CHANG)

  • 정주열;이현
    • 혜화의학회지
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    • 제13권1호
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    • pp.243-254
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    • 2004
  • Objectives & Methods : This study was to study etiology and pathologic mechanism and to study acupuncture and moxibustion treatment for GU-CHANG. I've got compared and analyzed 42 kinds of book since Nei Jing(內經). Results and Conclusion : 1. The etiologies are heat of heart and spleen, insufficiency of middle -warm energy, deficiency of yin lead to hyperactivity of fire. 2. The meridians of acupuncture points which were used much for GU-CHANG were Conception Vessel Meridian, Stomach Meridian and Bladder Meridian. 3. The frequency of using acupunture points in this treatment were Hapkok(LI4)-17th, Sungjang(CV24)-14th, Nogung(P8)-12th, Kumjin Okaeg(NH70)-11th.

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급성 연쇄상구균 감염후 사구체 신염에 동반된 고혈압성 뇌병증 3례의 MRI 소견 (Three Cases of Hypertensive Encephalopathy in Acute Poststreptococcal Glomerulonephritis: MRI Findings)

  • 김형노;윤성민;김종신;이창연
    • Childhood Kidney Diseases
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    • 제1권1호
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    • pp.73-78
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    • 1997
  • The hypertensive encephalopathy is one of sudden convulsive cause in patients with acute post-streptococcal glomerulonephritis, but commonly, after the episode we can observe only mild to moderately hypertension in APSGN at ER. So these remains some question of what causes the convulsion in patients with APSGN. In this article, we report 3 cases of convulsion in APSGN with only moderate hypertension, the etiology of which proved to be due to hypertensive encephalopathy by the brain MRI. We believe that the brain MRI is a very useful test to diagnose the etiology of convulsion in APSGN and if brain edema in occipital lobe is detected by brain MRI, the convulsion would be due to hypertensive encephalopathy and the prognosis will be very good.

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