• 제목/요약/키워드: cellular growth

검색결과 1,490건 처리시간 0.029초

무선 인터넷 기반의 급액관리 시스템 (Irrigation System Based on Wireless Internet)

  • 조태경
    • 전기학회논문지P
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    • 제58권4호
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    • pp.422-426
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    • 2009
  • The key point on water culture is the precise irrigation control according to the growth condition of growing plant. And most existing irrigation controllers are operated independently. So, the manager must be resided at hydroponic farm. In this paper, we design the irrigation control system based on wireless Internet that can be providing various control functions according to the growth condition of growing plant by using the cellular phone.

Multiple Roles of Phospholipase D in Growth Factor Signaling

  • Ryu, Sung-Ho
    • 대한약학회:학술대회논문집
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    • 대한약학회 2003년도 Proceedings of the Convention of the Pharmaceutical Society of Korea Vol.1
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    • pp.107-108
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    • 2003
  • The epidermal growth factor (EGF) is an important signaling ligand for the mitogenesis of many cells. The EGF receptors use signaling molecule multicomplexes and dynamic protein networks for the transmission and amplification of the signals as well as for the regulation of the cellular responses. EGF signaling has been reported to be enhanced in various tumors by the overexpressed EGF receptor and/or the mediators such as phospholipase C-$\gamma$1(PLC$\gamma$1). (omitted)

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수지자석 제조기법과 전자파 흡수체 및 식물 성장촉진기의 개발 (Development of manufacturing method of resin magnet. plant growth promote machine and electro wave absorbent material)

  • 이오걸
    • 대한전기학회:학술대회논문집
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    • 대한전기학회 2000년도 학술대회 논문집 전문대학교육위원
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    • pp.13-16
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    • 2000
  • This paper developed a manufacturing method for resin magnet contained rare earth element and synthetic resin. Use this manufacturing method, developed a electro wave absorbent material and plant growth promote machine. Result of experiments shown to have excellent on bean sprouts cultivate, computer monitor, television monitor and cellular phone etc.

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TGF-$\beta$ INDUCES INVASIVE PHENOTYPE OF MCF10A HUMAN BREAST EPITHELIAL CELLS

  • Kim, Mi-Sung;Aree Moon
    • 한국독성학회:학술대회논문집
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    • 한국독성학회 2002년도 Molecular and Cellular Response to Toxic Substances
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    • pp.141-141
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    • 2002
  • Transforming growth factor-${\beta}$ (TGF-${\beta}$), a hormonally active polypeptide found in normal and transformed tissues, regulates cellular growth and phenotyphic plasticity. We have previously shown that H-ras, but not N-ras, induces invasive phenotype in MCF10A human breast epithelial cells.(omitted)

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Dynamics of Pore Growth in Membranes and Membrane Stability

  • W. Sung;Park, P. J.
    • 한국생물물리학회:학술대회논문집
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    • 한국생물물리학회 1998년도 학술발표회
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    • pp.15-15
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    • 1998
  • Pores can form and grow in biomembranes because of factors such as thermal fluctuation, transmembrane electrical potential, and cellular environment. We propose a new statistical physics model of the pore growth treated as a non-Markovian stochastic process, with a free energy barrier and memory friction from the membrane matrix treated as a quasi-two-dimensional viscoelastic and dielectric fluid continuum.(omitted)

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Testosterone이 토끼 근위 세뇨관 상피세포의 성장에 미치는 영향 (Effect to Testosterone on the Growth of Primary Rabbit Proximal Tubule Cells in Serum-Free Medium)

  • 추민호;박승준;정주호;정지창
    • 대한약리학회지
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    • 제31권1호
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    • pp.85-93
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    • 1995
  • Testosterone이 serum-free medium에서 배양한 토끼의 신장 근위세뇨관 상피세포의 세포성장과 기능에 미치는 영향을 관찰한 바 다음과 같은 결과를 얻었다. 1. 토끼의 신장 근위세뇨관 상피세포는 testosterone 1 nM의 농도에서 유의한 세포 성장 촉진 효과를 나타내었고, testosterone 10 nM이상의 농도에서는 세포성장이 억제되었다. 2. Testosterone은 serum-free medium에서 성장촉진인자의 하나인 hydrocortisone을 growth supplement로 넣어준 serum-free medium에서 토끼 신장의 근위세뇨관 상피세포의 성장을 촉진시키었다. 3. Testosterone은 hydrocortisone을 growth supplement로 넣어준 serum-free medium에서 토끼 신장의 근위세뇨관 상피세포의 성장을 촉진시키었다. 4. Testosterone은 Northern blot analysis에 의하여 확인한 토끼 신장의 근위 세뇨관 상피세포의 ${\beta}-actin$ mRNA level은 증가되었다. 이상의 결과로 미루어 보아, serum-free 그리고 hormonally defined media에서 testosterone이 토끼의 신장 근위세뇨관 상피세포의 성장 및 기능에 대하여 촉진적으로 작용하는 것은 cellular mecrofilament의 중요한 구성단백의 하나로 밝혀진 ${\beta}-actin$의 합성 증가에 기인하는 것으로 생각된다.

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Hippo Signaling Circuit and Divergent Tissue Growth in Mammalian Eye

  • Moon, Kyeong Hwan;Kim, Jin Woo
    • Molecules and Cells
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    • 제41권4호
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    • pp.257-263
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    • 2018
  • Vertebrate organ development is accompanied by demarcation of tissue compartments, which grow coordinately with their neighbors. Hence, perturbing the coordinative growth of neighboring tissue compartments frequently results in organ malformation. The growth of tissue compartments is regulated by multiple intercellular and intracellular signaling pathways, including the Hippo signaling pathway that limits the growth of various organs. In the optic neuroepithelial continuum, which is partitioned into the retina, retinal pigment epithelium (RPE) and ciliary margin (CM) during eye development, the Hippo signaling activity operates differentially, as it does in many tissues. In this review, we summarize recent studies that have explored the relationship between the Hippo signaling pathway and growth of optic neuroepithelial compartments. We will focus particularly on the roles of a tumor suppressor, neurofibromin 2 (NF2), whose expression is not only dependent on compartment-specific transcription factors, but is also subject to regulation by a Hippo-Yap feedback signaling circuit.

자외선 형광장치현미경으로 본 화분관생장과 석탄 및 aureomycin의 상호작용에 대하여 (On the synergistic action of calcium and antibiotics in pollen growth as observed with ultraviolet fluorescence microscopy)

  • 곽병환
    • Journal of Plant Biology
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    • 제10권3_4호
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    • pp.1-3
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    • 1967
  • Observations were made on Crinum and Catharanthus pollen growth in artificial media by an ultraviolet transmission fluorescence microscope showing synergistic effect on pollen growth with calcium (Ca) and aureomycin. Bright yellow fluorescence of aureomycin enabling to trace out at tissue or cellular level did reveal that the greater accumulation of fluorescence occurred in the pollen tube wall if Ca was supplemented to the media than when aureomycin alone was present. The promotive pollen growth the media of Ca alone was further enhanced by the addtion of aureomycin. It was assumed that the promoted pollen growth with aureomycin in the Ca media was probably brought about by a supporting role of aureomycin in the Ca action.

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PIF4 Integrates Multiple Environmental and Hormonal Signals for Plant Growth Regulation in Arabidopsis

  • Choi, Hyunmo;Oh, Eunkyoo
    • Molecules and Cells
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    • 제39권8호
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    • pp.587-593
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    • 2016
  • As sessile organisms, plants must be able to adapt to the environment. Plants respond to the environment by adjusting their growth and development, which is mediated by sophisticated signaling networks that integrate multiple environmental and endogenous signals. Recently, increasing evidence has shown that a bHLH transcription factor PIF4 plays a major role in the multiple signal integration for plant growth regulation. PIF4 is a positive regulator in cell elongation and its activity is regulated by various environmental signals, including light and temperature, and hormonal signals, including auxin, gibberellic acid and brassinosteroid, both transcriptionally and post-translationally. Moreover, recent studies have shown that the circadian clock and metabolic status regulate endogenous PIF4 level. The PIF4 transcription factor cooperatively regulates the target genes involved in cell elongation with hormone-regulated transcription factors. Therefore, PIF4 is a key integrator of multiple signaling pathways, which optimizes growth in the environment. This review will discuss our current understanding of the PIF4-mediated signaling networks that control plant growth.

Fibroblast Growth Factor Receptor 3 (FGFR3) Signaling in Achondroplasia

  • Park, Sung Won
    • Journal of mucopolysaccharidosis and rare diseases
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    • 제2권2호
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    • pp.46-49
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    • 2016
  • Achondroplasia is autosomal dominant genetic disease and fibroblast growth factor receptor 3 (FGFR3) is currently known to be the only gene that causes achondroplasia. Gain-of function mutation in fibroblast-growth-factor-receptor 3 (FGFR3) causes the disease and C-type natriuretic peptide (CNP) antagonizes FGFR3 downstream signaling by inhibiting the pathway of mitogen-activated protein kinase (MAPK). As FGFR3-related skeletal dysplasias are caused by growth attenuation of the cartilage, chondrocytes appear to be unique in their response to FGFR3 activation. However, the full spectrum of molecular events by which FGFR3 mediates its signaling is just beginning to emerge. This article summaries the mechanisms of FGFR3 function in skeletal dysplasias, the extraordinary cellular manifestations of FGFR3 signaling in chondrocytes, and finally, the progress toward therapy for ACH.