• Title/Summary/Keyword: Toxicosis

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A study on fetal toxicosis removal therapy (하태독법(下胎毒法)에 대한 문헌적(文獻的) 고찰(考察))

  • Chang, Gyu-Tae;Kim, Jang-Hyun;Kang, Mi-Sun
    • The Journal of Pediatrics of Korean Medicine
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    • v.17 no.1
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    • pp.29-51
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    • 2003
  • This study was progressed for oriental medical care of neonates by fetal toxicosis removal therapy, and the results as follows: 1. The purpose of fetal toxicosis removal therapy : It removes dirty blood in neonatal mouth and prevents diseases. 2. Kinds of fetal toxicosis removal therapy : Radix Glycyrrhizae(甘草), Rhizoma Coptidis(黃連), Cinnabaris-Mel(朱蜜), Calculus Bovis(牛黃), Sojae Semen Praepartum, Juice of Semen Allii Tuberosi(非汁) and mercury(汞粉). 3. A period of fetal toxicosis removal therapy : At birth, 3rd day or so, one week or so, after umbilical omission. 4. Methods of fetal toxicosis removal therapy: Purgative therapy(下法) and Emetic therapy(吐法).

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A clinical case report of Hyperthyroidism(T3 toxicosis) patient (갑상선기능항진증(T3 중독증) 치험 1례에 대한 보고)

  • Lee, Ju-Ho;Choi, Jung-Sik;Cho, Chung-Sik;Kim, Cheol-Jung
    • Journal of Haehwa Medicine
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    • v.16 no.2
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    • pp.225-228
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    • 2007
  • Objective : This study was designed to evaluate oriental medicine therapy on a hyperthyroidism(T3 toxicosis) patient Methods : The clinical data was analyzed on a patient with hyperthiroidism(T3 toxicosis). This patient was treated with oriental medicine therapy only. After treatment, we analyzed a paient clinical symptom and thyroid function(T3, free T4, TSH) Results : After treatment, improvement was seen in clinical symptom, thyroid function test. conclusion : The study suggests that oriental medicine therapy is effective on the treatment of hyperthyroidism(T3 toxicosis).

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A Study on Bu一ja(<附子>, Oh-du<烏頭>) Toxicosis (부자(附子)오두(烏頭) 중독증(中毒症)에 관한 소고(小考))

  • Na, Chang-Su;Yun, Yeo-Chung
    • The Journal of Korean Medicine
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    • v.16 no.1 s.29
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    • pp.451-467
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    • 1995
  • In order to study on Bu-ja(Oh-du) toxicosis, the outhor investigated many documents. The results obtained were as follows: 1. Toxicosis of Bu-ja(Oh-du) arised mainly from carelessness in use. 2. In the symptoms of Bu-ja(Oh-du), Heart System(心系) symptom often came out. 3. The general and local paralysis, falling of blood pressure in Heart System(心系) symptom : vertigo, cramp in Liver System(肝系) symptom : nausea, vomiting in Spleen System (脾系) symptom : difficulty of breathing in Lung System(肺系) symptom : wasting disease in Kidney System (腎系) symptom frequently originated from toxicosis of Bu-ja(Oh-du). 4. In the detoification method, there are atropin injection, carrying out a gastric lavage, and supplying a liquid medicine from the western medicine : taking ginger-licorice root decoction, taking mung beans thick decoction and getting vomited with soaked cinnamon from the oriental medicine. And also keeping warm and oxygen inhalation were used to counteract poison. 5. A pregnant woman, heat of excess type(實熱), yang sydrome(陽證 )must not be taken Bu-ja(Oh-du).

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A Study of Computer Toxicosis and Deviant Behavior in Vocational High School Students (실업계 고교생의 컴퓨터 중독과 일탈행동 실태 연구)

  • Choi, Nak-Kwoan;Chung, Jong-In
    • The Journal of Korean Association of Computer Education
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    • v.7 no.3
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    • pp.79-89
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    • 2004
  • Social problems are coming to light due to the dysfunction which is caused by the improvement of ICT and the increasing information with computers and one of these problems is teenagers' computer toxicosis. In order to measure such an toxicosis, test paper that is developed by Kimberly S. Young is typically used. In this study, along with the test paper, localized Korean Internet addiction test (K-index) was used and the study was also made on the basis of such result. With results of sorting the data on the basis of K-index diagnosis criteria, it showed general users were 534 (64.8%), with potential risk users were 229 (27.8%) and high risk users were 61 (7.4%). This study was for the purpose of finding out sufficient and essential conditions ( outer behaviors ) of computer toxicosis for high risk users and searching for preventive measures for them.

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Chronic Ovine Copper Toxicosis in Korea (국내에서 발생한 면양의 만성 동중독증 예의 관찰)

  • 김대용;권오경;서일복
    • Journal of Veterinary Clinics
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    • v.15 no.2
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    • pp.455-459
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    • 1998
  • Ten sheeps from one farm had sudden onset of anorexia, jaundice and hemoglobinuria and died within 3 days after showing clinical signs during 3 months period. Postmortom examination was performed on one case and copper concentrations in the livers kidney and serum of the necmpsied minim were analysed. Grossly, the conjunctive, subcutaneous tissue and abdominal fat were generally icteric. The liver was enlarged with yellowish orange in color. The kidney was enlarged with dark red in color and the urinary bladder was filled with dark red urine. Histopathologically, centrilobular hepatocellular necrosis, neutrophilic infiltrations bile stasis and aggregation of fine granules-laden macrophages in the portal area were noted in the liver. Most of the Bowman's spaces and renal tubules were filled with homogenous eosinophilic fluid. Extramedullary hematopoiesis was noted in the submandibular lymph node. Copper concentrations in serum, liver and kidney of the necropsied animal were 25.0, 2732.8 and 471.3 ppm respectively. Based on the gross and histopathological findings and the high copper concentrations in the organs, this case was diagnosed as chronic copper toxicosis. Possible etiology on this outbreak is also discussed.

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Toxicity of ivermectin in Jindo-dogs 1. Clinical and hematological observation (진돗개에서 ivermectin의 독성 I. 임상증상과 혈액학적 변화 관찰)

  • Lee, Chai-yong;Oh, Seok-il;Lee, Chung-gil
    • Korean Journal of Veterinary Research
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    • v.37 no.4
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    • pp.855-862
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    • 1997
  • Ivermectin is a synthetic derivative of the naturally occurring avermectin $B_{1a}$ (22, 23-dihydroavermectin $B_{1a}$) and $B_{1b}$ (22, 23-dihydroavermectin $B_{1b}$), It is widely used as antiparasitic and pesticidal agents because of its remarkably potent and broad spectrum of antiparasitic activity. Although the drug has shown excellent anthelmintic effects, development of toxicosis in some animals such as the Collie species of dog is well documented. However, no studies have been reported on the toxic effects of the drug in Korean native animals such as the Jindo dog. The toxic effect of ivermectin was evaluated in 25 Jindo dogs divided into five groups which were orally administered with ivermectin at dosage levels of $200{\mu}g/kg$, $300{\mu}g/kg$, $600{\mu}g/kg$ and $2,500{\mu}g/kg$ of body weight, respectively. Toxic signs were not observed in the groups receiving $200{\mu}g/kg$ and $300{\mu}g/kg$ B.W. ivermectin. One dog developed mild clinical signs of toxicosis in the group receiving $600{\mu}g/kg$ dosage of ivermectin. In the group with $2,500{\mu}g/kg$ dosage, all dogs developed mild (salivation, drooling, vomiting, mydriasis, and/or confusion) and/or moderate (ataxia and tremors) clinical signs of toxicosis. Hematologic changes were not observed in the groups receiving $200{\mu}g/kg$, $300{\mu}g/kg$ and $600{\mu}g/kg$ B.W. ivermectin. In the groups receiving $2,500{\mu}g/kg$ B.W., total erythrocyte counts, total and differential leukocyte counts and hemoglobin levels were not affected by drug. Aspartate aminotransferase levels were increased after administration of ivermectin, while serum cholesterol and blood glucose levels were decreased.

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A case of treatment on amitraz toxicosis in a Thoroughbred racehorse (Thoroughbred 경주마에서 amitraz 중독증 치료 1례)

  • Yang, Jaehyuk;Song, Heeeun;Lee, Kyuong-Kap;Jee, Youngheun;Woo, Ho-Choon;Lim, Yoon-Kyu
    • Korean Journal of Veterinary Research
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    • v.50 no.3
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    • pp.253-257
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    • 2010
  • A 3-year-old female Thoroughbred racehorse was presented following the accidental oral and skin administration of amitraz. This case report describes the clinical signs and the treatment of this horse. Clinical signs of amitraz toxicosis are associated with the stimulation of alpha2-adrenergic receptors. Amitraz is seldom fatal because the effects can be reversed by alpha2-adrenergic antagonists. The horse displayed typical clinical signs of colic, including pawing, small hard drops, tranquillisation, depression, ataxia, muscular incoordination and impaction colic lasting up to 7 days. The syndrome was accompanied by mild dehydration. The horse survived after persistent symptomatic treatment, including the giving of intravenous fluids, antibiotics, multiple doses of mineral oil per os, nonsteroidal anti-inflammatory drugs and dexamethasone intramuscularly and intravenously.

Ethylene glycol toxicosis in a dog (개의 ethylene glycol 중독)

  • Jean, Young-Hwa;Woo, Gye-Hyeong;Kim, Jae-Hoon;Hwang, Eui-Kyung;Seo, Il-Bok
    • Korean Journal of Veterinary Pathology
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    • v.2 no.2
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    • pp.147-151
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    • 1998
  • A five-month old female Great Dane with a history of depression and convulsion was submitted to the Pathology and Diagnosis Reference Division of National Veterinary Research and Quarantine service. The dog had ingested concentrated antifreeze and died 12 days after the sudden onset of clinical signs. Main gross lesion was limited to swollen kidneys with diffusely fine granularity on the cut surface. Principle microscopic lesions were focused on the kidneys including dilated Bowman's space, thickened glomerular basement membrane, many light yellowish crystals, and proteinaceous casts in some tubules. Some neutrophils and lymphocytes were focally infiltrated in interstitium of the cortex and medulla. Crystals were angular or quite irregular in shape and arranged in sheave or rosette form. Under polarized light, the crystals were birefringent, which is typical of calcium oxalate crystals. Crystals were more concentrated in the cortex than in the medulla. A few crystals were also observed in the small blood vessels of the brain. Based upon the history of ingestion of antifreeze and the identification of calcium oxalate crystals in the tissue sections of the kidney and brain, this case was diagnosed as ethylene glycol toxicosis in a dog.

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