• Transactions of the Korean Society of Mechanical Engineers B
• /
• v.20 no.3
• /
• pp.1114-1122
• /
• 1996

Variation of temperature field in a Hele-Shaw convection cell was measured by using a HSI true color image processing system and TLC(Thermochromic Liquid Crystal) solution. The relationship between the hue value of TLC color image and real temperature was obtained and this calibration result was used to measure the true temperature. The temperature field in the Hele-Shaw convection cell shows periodic characteristics of 45 sec at Ra = 9.3 * 10$\^$6/. The temperature field measurement technique developed in this study was proved to be a useful and powerful tool for analyzing the unsteady thermal fluid flows.

• Transactions of the Korean Society of Mechanical Engineers B
• /
• v.21 no.10
• /
• pp.1254-1261
• /
• 1997

Pulse boiling, the unsteady periodic boiling phenomenon appearing in the evaporator of thermosyphons was investigated by many researchers. In the present study investigations were conducted to examine the evolution of flow patterns at the evaporator, and changes in thermodynamic state that each of liquid pool and vapor experiences through 1 cycle of pulse boiling process. For wall and liquid pool the degree of superheat for the onset of nucleation was examined. It revealed that the degree of superheat increased with the increase of pulse period, reaching to 16.5 deg.C and 23 deg.C for liquid pool and evaporator wall respectively at .tau.=80 sec. The data on flow patterns obtained through series of operation tests were plotted in the coordinates of heat flux and vapor pressure to get a regime map. Further this map could be used to figure out the conditions of pulse boiling for a thermosyphon.

• Journal of the Korean Society for Aeronautical & Space Sciences
• /
• v.46 no.5
• /
• pp.368-375
• /
• 2018

In the present study, aerodynamic load analysis for a floating off-shore wind turbine was conducted to examine the effect of periodic platform motion in the direction of 6-DOF on rotor aerodynamic performance. Blade-element momentum method(BEM) was used for a numerical simulation, the unsteady airload effects due to the flow separation and the shed wake were considered by adopting a dynamic stall model based on the indicial response method. Rotor induced downwash was estimated using the momentum theory, coupled with empirical corrections for the turbulent wake states. The periodic platform motions including the translational motion in the heave, sway and surge directions and the rotational motion in the roll, pitch and yaw directions were considered, and each platform motion was applied as a sinusoidal function. For the numerical simulation, NREL 5MW reference wind turbine was used as the target wind turbine. The results showed that among the translation modes, the surge motion has the largest influence on changing the rotor airloads, while the effect of pitch motion is predominant for the rotations.

• Transactions of the Korean Society of Mechanical Engineers B
• /
• v.40 no.8
• /
• pp.511-517
• /
• 2016

Three-dimensional models of stenosis blood vessels were prepared using a 3D printer. The models included a straight pipe with axisymmetric stenosis and a pipe that was bent $10^{\circ}$ from the center of stenosis. A refractive index matching method was utilized to measure accurate velocity fields inside the 3D tubes. Three different pulsatile flows were generated and controlled by changing the rotational speed frequency of the peristaltic pump. Unsteady velocity fields were measured by a time-resolved particle image velocimetry method. Periodic shedding of vortices occurred and moves depended on the maximum velocity region. The sizes and the positions of the vortices and symmetry are influenced by mean Reynolds number and tube geometry. In the case of the bent pipe, a recirculation zone observed at the post-stenosis could explain the possibility of blood clot formation and blood clot adhesion in view of hemodynamics.

• Sleep Medicine and Psychophysiology
• /
• v.6 no.1
• /
• pp.19-25
• /
• 1999

Sleep alters both breathing pattern and the ventilatory responses to external stimuli. These changes during sleep permit the development or aggravation of sleep-related hypoxemia in patients with respiratory disease and contribute to the pathogenesis of apneas in patients with the sleep apnea syndrome. Fundamental effects of sleep on the ventilatory control system are 1) removal of wakefulness input to the upper airway leading to the increase in upper airway resistance, 2) loss of wakefulness drive to the respiratory pump, 3) compromise of protective respiratory reflexes, and 4) additional sleep-induced compromise of ventilatory control initiated by reduced functional residual capacity on supine position assumed in sleep, decreased $CO_2$ production during sleep, and increased cerebral blood flow in especially rapid eye movement(REM) sleep. These effects resulted in periodic breathing during unsteady non-rapid eye movement(NREM) sleep even in normal subjects, regular but low ventilation during steady NREM sleep, and irregular breathing during REM sleep. Sleep-induced breathing instabilities are divided due primarily to transient increase in upper airway resistance and those that involve overshoots and undershoots in neural feedback mechanisms regulating the timing and/or amplitude of respiratory output. Following ventilatory overshoots, breathing stability will be maintained if excitatory short-term potentiation is the prevailing influence. On the other hand, apnea and hypopnea will occur if inhibitory mechanisms dominate following the ventilatory overshoot. These inhibitory mechanisms include 1) hypocapnia, 2) inhibitory effect from lung stretch, 3) baroreceptor stimulation, 4) upper airway mechanoreceptor reflexes, 5) central depression by hypoxia, and 6) central system inertia. While the respiratory control system functions well during wakefulness, the control of breathing is commonly disrupted during sleep. These changes in respiratory control resulting in breathing instability during sleep are related with the pathophysiologic mechanisms of obstructive and/or central apnea, and have the therapeutic implications for nocturnal hypoventilation in patients with chronic obstructive pulmonary disease or alveolar hypoventilation syndrome.