• Design & Manufacturing
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• 제2권4호
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• pp.11-15
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• 2008

In the sheet metal forming using a high speed press machine, driving device, such as crank, link, and knuckle mechanism, has to be designed in consideration of impact at a moment when press die contact with material, because the impact affects a dimensional accuracy of products and a life span of press die. In this study, dynamic analysis was performed using numerical simulation in order to verify the impact reduction effect for proposed double knuckle mechanism by estimating rolling and pitching moment of slide.

• 한국소성가공학회:학술대회논문집
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• 한국소성가공학회 2008년도 춘계학술대회 논문집
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• pp.243-246
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• 2008

The press machine is actuated by the rotating motion of crank shaft and the reciprocating motion of slide. In recent years, unbalance moments and forces to the main frame attract many researches, as press technology becomes more miniaturized, precise, and rapid. In order to control vibrations caused by the rapid motions of the crank shaft and slide, this paper studies a resolution reducing the unbalance at the high speed knuckle press.

• 한국자동차공학회논문집
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• 제21권1호
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• pp.121-127
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• 2013

In this study, suspension geometry is controlled to improve vehicle handling performance. The toe and camber of the rear suspension is controlled independently by using a double knuckle structure designed to enhance the vehicle cornering stability. Camber and toe changes in the rear wheel during high speed turning maneuver are important factors that influence the vehicle stability. Toe in the rear outer wheel plays a dominant role in cornering. A control algorithm for the camber and the toe angle input is developed to carry out the control simulation of the vehicle such as single lane change, the steady state cornering, the double lane change and the step steering simulation. Effects of the camber and toe angle control are analyzed from the computer simulations. A double lane change simulation revealed that the suspension mechanism with variable camber angle and variable toe angle decreases the peak body slip angle and peak yaw rate, 50% and 10%, respectively.

• 소성∙가공
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• 제17권8호
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• pp.643-648
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• 2008

Ultra-thin sheet metal forming techniques are required in precision forming of miniaturized and integrated products. In order to manufacture a good quality and low cost ultra-thin sheet metal products, a highly precise high-speed press is needed. The precision of a press is related with its vibration characteristics during pressing operation. This study evaluated the vibration characteristics of a proposed press design using computer simulation. The analysis compares the static deformation characteristics of the slide and the slide motion for the metal forming of an ultra-thin sheet of thickness less than 0.1mm. Further, in order to minimize the vibrations during high speed pressing operation, revolution balances of the eccentric shaft and the balance weight device is also considered. Finally, modal analysis is used to characterize the natural frequency of vibration of the press.

• Genomics & Informatics
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• 제8권2호
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• pp.70-75
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• 2010

Asp66his, Asp54Lys, and Asp50Asn are mutations in connexin 26 that are observed in the clinic and give rise to autosomal dominant syndromes. They are the result of point mutations in the human gap junction ${\beta}-2$ gene. In order to investigate the structural mechanism of Bart-Pumphrey Syndrome, Keratitis-Ichthyosis-Deafness Syndrome, and Vohwinkel Syndrome, homology modeling was carried out. Asp66 has direct contact with Asn62 by two hydrogen bonds in the wild-type protein, and in Asp66His, the biggest change observed is a tremendous energy increase caused by hydrogen bond breakage to Asn62. Shifts in the side chain and new hydrogen bond formation are observed for Lys54 compared to the wild-type protein (Asn54) and result in closer contact to Val84. Asp50Asn causes a significant decrease in bond energy, and residual charge reversal repels the ion and metabolites and, hence, inhibits their transportation. Such perturbations are likely to be a factor contributing to abnormal functioning of ion channels, resulting cell death and disease.