• The Transactions of the Korean Institute of Electrical Engineers B
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• v.50 no.8
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• pp.392-401
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• 2001

This paper presents the single-stage high power factor AC to DC converter operated in active-clamp mode. The proposed converter is added active-clamping circuit to boost-flyback single-stage power factor corrected power supply. The active-clamping circuit limits voltage spikes, recycles the energy trapped in the leakage inductance, and provides a mechanism for achieving soft switching of the electronic switches to reduce the switching loss. The auxiliary switch of active-clamping circuit uses the same control and driver circuit as the main switch to reduce the additional cost and size. To verify the performance of the proposed converter, a 100W converter has been designed. The proposed converter gives good power factor correction, low line current harmonic distortions, and tight output voltage regulation, as used unity power factor.

• Journal of the Korea institute for structural maintenance and inspection
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• v.10 no.1
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• pp.131-138
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• 2006

Structural bracing concept equipped with a new and efficient friction based energy dissipation device is referred to Friction SliP Brace (FSB) where the behavior of the brace components is elastic until the axial resistant force in the brace exceeds the friction force developed at the frictional interface of the device. In this study, the FSB concept is modified and new type of hybrid energy dissipation device, the Active Friction SliP Braces (AFSB), is described. The FSB is by far improved in the AFSB by inclusion of an active clamping mechanism on the friction interface. The clamping action regulated by the developed algorithm is altered during the response of the building. The results indicate that the action of dissipating vibrational energy in the AFSB impacts on the response at later cycles by keeping the drift amplitudes at much lower levels, revealing overshooting problem due to its early slippage. Providing predetermined constant incremental strengths to the building by AFSB medium improves response by reducing drift amplitudes and base shear under small and medium amplitude ground accelerations.

• Journal of Pharmaceutical Investigation
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• v.2 no.1
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• pp.18-30
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• 1972

Renal pathways for excretion of sulfadiazine has been studied by standard clearance technique in the rabbit. 1. Large part of sulfadiazine filtered in the glomeruli is reabsorbed in the tubules, as visualized from the fact that Csd (clearance of sulfadiazine) amounts only a fraction of simultaneously measured Ccr (GFR). 2. Csd changed linearly with the rate of urine flow, whether it is increased by the duir etics or decreased by clamping u reter. 3. Csd remained unchanged until the plasma level of the Csdremained unchanged drug reached 10.0 mg%, and the amount transported in the tubules increased linearly with the increase in the load, exhibiting No maximum capacity for transport. 4. Csd was increased by 2,4-dinitrophenol which is an uncoupling agent of oxidative phosphorylation and decreased by probenecid which is on uricosuric agent. 5. During sodium bicarbonate infusion net secretion of sulfadiazine by tubules observed. All the evidences obtained in the rabbit indicated that sulfadiazine was reabsorbed by active, energy-requiring, or passive, simple diffusion, process, and secreted simultaneously by a probenecid-sensitive, active procss.

• The Korean Journal of Physiology
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• v.25 no.2
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• pp.171-177
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• 1991

This study was carried out to determine effect of acute renal ischemia on transport function of organic cation, tetraethylammonium (TEA), in rabbit kidney proximal tubule. Clamping of the renal artery for 30 and 60 min produced a polyuria which was accompanied by an increase in $Na^+$ excretion. The capacity of kidney cortical slices to accumulate TEA was increased after 30 and 60 min of ischemia. When blood flow was restored for 30 min after 30 and 60 min of ischemia, the augmented TEA uptake was recovered to the control values. Oxygen consumption of cortical slices was stimulated after 30 min of ischemia, whereas it was not altered by 60 min of ischemia. A 90-min ischemia produced a significant inhibition of TEA uptake and tissue oxygen consumption. These results suggest that the basolateral transport system for organic cation persists after ischemic periods of 60 min despite evidence that tubular reabsorptive mechanism of $Na^+$ and water is markedly impaired. This may indicate that the active secretory systems of proximal tubule are more resistant to ischemic injury than the reabsorptive systems.