• 한국전자현미경학회:학술대회논문집
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• 한국현미경학회 2003년도 제34차 춘계학술대회
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• pp.31-33
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• 2003

• 제어로봇시스템학회:학술대회논문집
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• 제어로봇시스템학회 2002년도 ICCAS
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• pp.85.4-85
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• 2002

$\textbullet$ Flying Robot is a kind of UAV as an autonomous hovering platform. $\textbullet$ Control system is high complex, and non-linear Multiple-Input, Multiple-Output (MIMO) system. $\textbullet$ Eexperiment-device capable of measuring roll, yaw and pitch angle with PID controller by PC. $\textbullet$ This paper proves results of simulation through velocity control in condition.

• 한국마린엔지니어링학회:학술대회논문집
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• 한국마린엔지니어링학회 2005년도 후기학술대회논문집
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• pp.202-203
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• 2005

we present a pressure sensor based on the mechanically formed long-period fiber gratings. The attenuation properties of an output power as a function of an external pressure is able to apply to the in-line fiber pressure sensors for the vessels.

• 한국독성학회:학술대회논문집
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• 한국독성학회 2005년도 춘계 국제심포지엄 및 학술대회
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• pp.127-128
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• 2005

• 해양환경안전학회:학술대회논문집
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• 해양환경안전학회 2017년도 공동학술발표회
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• pp.27-28
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• 2017

• 산업공학
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• 제6권2호
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• pp.67-78
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• 1993

In this paper, we deal with the application of the fuzzy sets theory to evaluate and estimate the system reliability under the fault tree analysis. We formulate the uncertainty of component reliability to fuzzy sets, and propose a procedure for obtaining the system reliability in case the system structure is described by fault tree. An importance measure of each component is proposed. Computer program for fuzzy fault tree analysis(FFTA) is developed using C language to obtain the system reliability and the component‘s fuzzy importance.

• 한국자기학회:학술대회 개요집
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• 한국자기학회 2008년도 Asian Magnetics Conference
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• pp.159.2-159.2
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• 2008

• 대한전기학회:학술대회논문집
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• 대한전기학회 1998년도 하계학술대회 논문집 F
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• pp.2139-2141
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• 1998

This study was carried out about the manufacture of test equipment for traction Motor Driving. The power converter is driven by two converter serially and keep a high power factor of power source. This system is haven all the characteristic of voltage source converter by having a processing ability of regenerating power. The traction motor is controlled by IGBT inverter. The test equipment is composed of traction motor, torque-meter, clutch, and inertia body of tubular type.

• The Korean Journal of Physiology and Pharmacology
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• 제21권3호
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• pp.327-334
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• 2017

Epidemiologic interest in particulate matter (PM) is growing particularly because of its impact of respiratory health. It has been elucidated that PM evoked inflammatory signal in pulmonary epithelia. However, it has not been established $Ca^{2+}$ signaling mechanisms involved in acute PM-derived signaling in pulmonary fibroblasts. In the present study, we explored dust particles PM modulated intracellular $Ca^{2+}$ signaling and sought to provide a therapeutic strategy by antagonizing PM-induced intracellular $Ca^{2+}$ signaling in human lung fibroblasts MRC5 cells. We demonstrated that PM10, less than $10{\mu}m$, induced intracellular $Ca^{2+}$ signaling, which was mediated by extracellular $Ca^{2+}$. The PM10-mediated intracellular $Ca^{2+}$ signaling was attenuated by antioxidants, phospholipase blockers, polyADPR polymerase 1 inhibitor, and transient receptor potential melastatin 2 (TRPM2) inhibitors. In addition, PM-mediated increases in reactive oxygen species were attenuated by TRPM2 blockers, clotrimazole (CLZ) and N-(p-amylcinnamoyl) anthranilic acid (ACA). Our results showed that PM10 enhanced reactive oxygen species signal by measuring DCF fluorescence and the DCF signal attenuated by both TRPM2 blockers CLZ and ACA. Here, we suggest functional inhibition of TRPM2 channels as a potential therapeutic strategy for modulation of dust particle-mediated signaling and oxidative stress accompanying lung diseases.

• Journal of Microbiology and Biotechnology
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• 제25권7호
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• pp.1047-1055
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• 2015

For the search of a potent first-in-class compound to inactivate macrophages responsible for inflammatory responses, in the present study, we investigated the anti-nflammatory effects of YH-1118, a novel synthetic compound, in a lipopolysaccharide (LPS)-stimulated mouse macrophage cell line, Raw 264.7. YH-1118 inhibited LPS-induced nitric oxide (NO) production and inducible NO synthase (iNOS) expression at both the protein and mRNA levels. The suppression of LPS-induced iNOS expression by YH-1118 was mediated via nuclear factor kappa B (NF-κB), but not activator protein-1 (AP-1) transcription factor. This was supported by the finding that YH-1118 attenuated the phosphorylation of inhibitor of κBα (IκBα) and nuclear translocation and DNA binding activity of NF-κB. Through the mechanisms that YH-1118 inhibited the activation of IκB kinases (IKKs), upstream activators of NF-κB, or p38 MAPK, YH-1118 significantly suppressed LPS-induced production of pro-inflammatory cytokines, tumor necrosis factor-α, interleukin-1β (IL-1β), and IL-6 (p < 0.05). In conclusion, our results suggest that YH-1118 inhibits LPS-induced inflammatory responses by blocking IKK and NF-κB activation in macrophages, and may be a therapeutic candidate for the treatment of various inflammatory diseases.