• Title/Summary/Keyword: T3 toxicosis

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A clinical case report of Hyperthyroidism(T3 toxicosis) patient (갑상선기능항진증(T3 중독증) 치험 1례에 대한 보고)

  • Lee, Ju-Ho;Choi, Jung-Sik;Cho, Chung-Sik;Kim, Cheol-Jung
    • Journal of Haehwa Medicine
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    • v.16 no.2
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    • pp.225-228
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    • 2007
  • Objective : This study was designed to evaluate oriental medicine therapy on a hyperthyroidism(T3 toxicosis) patient Methods : The clinical data was analyzed on a patient with hyperthiroidism(T3 toxicosis). This patient was treated with oriental medicine therapy only. After treatment, we analyzed a paient clinical symptom and thyroid function(T3, free T4, TSH) Results : After treatment, improvement was seen in clinical symptom, thyroid function test. conclusion : The study suggests that oriental medicine therapy is effective on the treatment of hyperthyroidism(T3 toxicosis).

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[ $T_3$ ] Toxicosis

  • Gang, Mun-Won;Kim, Jong-Sang;Park, Seong-Hak;Bang, Byeong-Gi;Choe, Yeong-Gil;Min, Byeong-Seok
    • 대한핵의학회:학술대회논문집
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    • 1976.06a
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    • pp.68.3-69
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    • 1976
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Experimental Trichothecene(T-2) Toxicosis in Korean Native Goats (한국 재래산양에서의 실험적 Trichothecene(T-2) 독소중독증)

  • Kim, Jong-shu
    • Korean Journal of Veterinary Research
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    • v.28 no.1
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    • pp.49-58
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    • 1988
  • To investigate the effects of T-2 toxin on the blastogenesis of lymphocytes, pathology, hemogram and blood chemistry in the goat, the korean native goats were treated orally with T-2 toxin for 21 days with a dosage of 0.6mg per kg body weight. The results were as follows: 1. The total count of leukocytes and lymphocytes decreased significantly from 14 to 21 days after treatment. 2. Mryeloid: erythroid ratios increased significantly on days 12 after treatment. 3. Delayed-type hypersensitivity skin reactions to tuberculin were reduced predominantly. 4. T-2 toxin induced prolonged prothrombin time. 5. Mitogenic responses of lymphocytes to both lipopolysaccharide and phytohemagglutinin were significantly depressed on days 7 and 14 after treatment. 6. Treatment of T-2 toxin caused marked depletion of lymphocytes in the thymus, mesenteric lymph node, peyer's patchs and spleen.

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