Salt injury in rice is caused mainly by the salinity in soil and in the irrigated water, and occasionaly by salinity delivered through typhoon from the sea. The salt concentration of rice plants increased with higher salinity in the soil of the rice growing. The climatic conditions, high temperature and solar radiation and dry conditions promote the salt absorption of rice plant in saline soil. The higher salt accumulation in the rice plant generally reduces the root activity and inhibits the absorption of minerals of rice plant, resulting the reduction of photosynthesis. The salt damages of rice plant, however, are different from different growth stage of rice plants as follows: 1. Germination of rice seed was slightly delayed up to 1.0% of salt concentration and remarkably at 1. 5%, but none of rice seeds were germinated at 2.5%. This may be due to the delayed water uptake of rice seeds and the inhibition of enzyme activity, 2. It was enable to establish rice seedlings at seed bed by 0.2% of salt concentration with some reduction of leaf elongation. The increasing of 0.3% salt concentration caused to the seedling death with varietal differences, but most of seedlings were death at 0.4% with no varietal differences. 3. Seedlings grown at the nursery over 0.1% salt, gradually reduced in rooting activity after transplanting according to increasing the salt concentration from 0.1% up to 0.3% of paddy field. However, the seedlings grown in normal seed bed showed no difference in rooting between varieties up to 0.1% but significantly different at 0.3% between varieties, but greatly reduced at 0.5% and died at last in paddy after transplanting. 4. At panicle initiation stage, rice plant delayed in heading by salt damage, at meiotic stage reduced in grains and its filling rate due to inhibition of glume and pollen developing, and salt damage at heading stage and till 3 weeks after heading caused to reduction of fertilization and ripening rate. In viewpoint of agricultural policy the overcoming strategy for salt injury is to secure sufficient water source. Irrigation and drainage systems as well as underground drainage is necessary to desalinize more effectively. This must be the most effective and positive way except cost. By cultural practice, growing the salt tolerant variety with high population could increase yield. The intermittent irrigation and fresh water flooding especially at transplanting and from panicle initiation to heading stage, the most sensitive to salt injury, is important to reduce the salt content in saline soil. During the off-cropping season, plough and rotavation with flooding followed by drainage, or submersion and drainage with groove could improve the desalinization. Increase of nitrogen fertilizer with more split application, and soil improvement by lime, organic matter and forign soil addition, could increase the rice yield. Shift of trans-planting is one of the way to escape from the salt injury.
Ji, Seon Yeong;Hwangbo, Hyun;Kim, Min Yeong;Kim, Da Hye;Park, Beom Su;Park, Joung-Hyun;Lee, Bae-Jin;Lee, Hyesook;Choi, Yung Hyun
Journal of Marine Bioscience and Biotechnology
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v.13
no.2
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pp.86-93
/
2021
A high salt diet contributes to kidney damage by causing hypoxia and oxidative stress. Recently, an increase in dietary salt has been reported to induce an inflammatory phenotype in immune cells, further contributing to kidney damage. However, studies on the exact mechanism and role of a high salt diet on the inflammatory response in the kidneys are still insufficient. In this study, a cisplatin-induced acute kidney injury model using C57BL/6 mice was used to analyze the effect of salt intake on kidney injury. Results showed that high salt administration aggravated kidney edema in mice induced by treatment with cisplatin. Moreover, the indicators of kidney and liver function impairment were significantly increased in the group cotreated with high salt compared with that treated with cisplatin alone. Furthermore, the exacerbation of kidney damage by high salt administration was also associated with a decrease in the number of cells in the immune regulatory system. Additionally, high salt administration further decreased renal perfusion functions along with increased cisplatin-induced damage to proximal tubules. This was accompanied by increased expression of T cell immunoglobulin, mucin domain 1 (a biomarker of kidney injury), and Bax (a pro-apoptotic factor). Moreover, cisplatin-induced expression of proinflammatory mediators and cytokines, including cyclooxygenase-2 and tumor necrosis factor-α in kidney tissue, was further increased by high salt intake. Therefore, these results indicate that the kidney's inflammatory response by high salt treatment can further promote kidney damage caused by various pathological factors.
Journal of the Korea Institute of Building Construction
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v.2
no.2
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pp.165-172
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2002
A reinforced concrete building neighboring in Pusan or Ulsan where is directly exposed to salt water contrasting with other in land areas contains much salt content percolated from the outside that the high salt content percolates and diffuses through the inside of reinforced concrete; therefore, an immovable tunic surrounding it begins to be destroyed and eroded with high speed. At the time, the cross-sectional area and volume expansion of re-bar reinforcing result in being cracks make a rapid progress gradually until they appear in the surface of the one, the phenomenon such as being a thin layer or falling off the part of it causes a lowering of its durability and might collapse the concrete construction. So far, we've investigated into salt content of reinforced concrete constructions neighboring in a seaside district and damage by carbonation, and we came to a conclusion as follows: $\circled1$ Under the oceanic circumstance a concrete construction is influenced by sea water directly that contains much amount of salt content contrasting with other constructions on inland areas. $\circled2$ Because of chloride penetration the carbonation of reinforced concrete made a rapid progress until more than the covering thickness of re-bar. $\circled3$ An old reinforced concrete building which has been piled up salt injury and proceeding the carbonation of its cross-sectional area. $\circled4$ According to rapidly cracking from the inside to surface of reiforced concrete, the phenomenon of being a thin layer or falling off the part of reinforced concrete results in a lowering of durability and shortening the life-time of concrete construction itself.
Objective : In the present study we analyzed neuroprotective and antiapoptotic effect of the difumarate salt S-15176, as an anti-ischemic, an antioxidant and a stabilizer of mitochondrial membrane in secondary damage following spinal cord injury (SCI) in a rat model. Methods : Three groups were performed with 30 Wistar rats; control (1), trauma (2), and a trauma+S-15176 (10 mg/kg i.p., dimethyl sulfoxide) treatment (3). SCI was performed at the thoracic level using the weight-drop technique. Spinal cord tissues were collected following intracardiac perfusion in 3rd and 7th days of posttrauma. Hematoxylin and eosin staining for histopatology, terminal deoxynucleotidyl transferase dUTP nick end labeling assay for apoptotic cells and immunohistochemistry for proapoptotic cytochrome-c, Bax and caspase 9 were performed to all groups. Functional recovery test were applied to each group in 3rd and 7th days following SCI. Results : In trauma group, edematous regions, diffuse hemorrhage, necrosis, leukocyte infiltration and severe degeneration in motor neurons were observed prominently in gray matter. The number of apoptotic cells was significantly higher (p<0.05) than control group. In the S-15176-treated groups, apoptotic cell number in 3rd and 7th days (p<0.001), also cytochrome-c (p<0.001), Bax (p<0.001) and caspase 9 immunoreactive cells (p<0.001) were significantly decreased in number compared to trauma groups. Hemorrhage and edema in the focal areas were also noticed in gray matter of treatment groups. Results of the locomotor test were significantly increased in treatment group (p<0.05) when compared to trauma groups. Conclusion : We suggest that difumarate salt S-15176 prevents mitochondrial pathways of apoptosis and protects spinal cord from secondary injury and helps to preserve motor function following SCI in rats.
The changes in contents of chlorophyll and free proline in the seedling leaves of ten rice cultivars as affected by salt stress were checked in order to obtain the basic information on the judgement of the degrees of salt injury. The difference in salt injury among the cultivars was clearly observed about 25 days after 6% salt treatment. Chlorophyll content was decreased in both Gayabyeo and Taebaegbyeo for 14 days after different salt treatment as salt concentration was increased and the decreased tendency was much higher in Taebaegbyeo than in Gayabyeo over 0.4% salt concentration. Chlorophyll content in Gayabyeo after 0.6% salt treatment was decreased slowly, while in Taebaegbyeo, deminished very rapidly as time progressed, therefore it decreased by about 16% in Gayabyeo and 67% in Taebaebyeo compared to the control at 20 days, respectively. The relationship between chlorophyll content and the degrees of salt injury in ten rice cultivars showed significant negative correlation at 10 day after 0.6% salt treatment. Free proline content in Gayabyeo was increased gradually for 14 days after different salt treatment as salt became higher, while in Taebaebyeo, it was increased rapidly under 0.6% but rather decreased under 0.8% salt concentration. Particularly, it was much higher Taebaegbyeo than in Gayabyeo under salt concentration from 0.4 to 0.6%. Free proline content in Gayabyeo after 0.6% salt treatment was increased from 15 days, on the other hand in Taebaegbyeo, it was increased from 5 days, but rather decreased from 20 days, and it was 6 times higher in Taebaegbyeo than in Gayabyeo at 10 days. There was significant positive correlation between free proline content and the degrees of salt injury in ten rice cultivars at 10 days after 0.6% salt treatment. From the above results, chlorophyll and free proline content may be used as an indicative character of intensity of salt stress as well as varietal difference in resistance to salt stress in the seedling stage.
This study was intended to investigate injury rates of landscape trees planted in the reclaimed seaside areas and to analyse their causes in planting environment. The planting environment such as soil properties, reclaimed depth of soil, soil hardness, salt consistency of soil, and drainage system were surveyed in 8 reclaimed seaside areas in the middle and southern regions of the country. Injury rates of 42 species, 1,233 trees and growth of branches in 6 species. 130 trees were measured and analysed to fond out salt-resistant trees in the reclaimed lands. The results of the survey are as follows; the average injury rate of evergreen trees was 32%. which was much lower than that of deciduous trees as 52%. The lower injured trees in the 8 reclaimed lands were Pinus thunbergii, Pinus parvinora. Juniperus chinensis 'Kaizuka', Pinus koraiensis, Acer trifdum, Koelreuteria paniculata and Metasequoia glyptostroboides. The higher injured trees were Acer palmatum, Magnolia kobus. Lagerstroemia indica, Diospiros kaki, Cercidiphyllum japonicum, Sorbus commixta, Prunus yedoensis, Pinus desinora, Chaenomeles sinensis, Albizzia julibrissin and Zelkowa serrata. At least the mounding of 1.7m depth of soil is needed from the tidal saline soil to the roots of trees to protect trees from salt injury Roots of trees were found to have infiltrated to 0.77m under the soil of which solidity was over 4.5kg/㎥. And 22 of total 25 soils were acid from pH 3.72 to pH 5.85. Injury rate of trees was much higher when they were detached from the sea, and planted on the drainage.
This study was conducted to evaluate the performance of 2,4-D amine salt (2,4-dichlorophenoxy acetic acid) 40EC in broadleaf weeds control under diverse conditions of rate and time of application and soil type in fall barley field. The results obtained were summarized as follows: 1) At the rate of 70cc prod./10a, 2,4-D amine salt treated during the fully tillered stage of barley was effective for the control of most broadleaf weeds without crop injury. 2) The fully tillered stage was a optimun time lot the application of 2,4-D amine salt in fall barley. Earlier(zero-to four-leaf stage) or late. (boot stage to flowering) application than this stage caused a crop injury and resulted in yield reduction. 3) It was possible to safely use 2,4-D amine salt in fall barley regardless of soil types if applied the rate of 70cc prod./10a at the fully tillered stage.
This study aimed to examine a suppression effect of salinity on extension rate of stem elongation of cucumber seedlings (Cucumis sativus L.) grafted with roots of figleaf gourd plant. The effects of application methods, timings, and concentrations of two salt types, sodium chloride (NaCl) and calcium chloride ($CaCl_2$), were compared to untreated control plants. In result, an obvious suppression effect on the excessive elongation of stem was obtained by both sub-irrigated and medium-mixed NaCl salt. An improvement in quality of transplants was also obtained by the sub-irrigated NaCl salt. Foliar-applied NaCl caused visible leaf injury when the concentration was higher than 40 mM; but, with no effect on suppressing the stem elongation. When the NaCl was applied at 7 days after grafting, a higher concentration of NaCl was demanded for suppressing the stem elongation compared to an application at the day of grafting. No effect of the NaCl salt on the fresh weights of 36-day grown plants was observed; but, there was a negative effect on the number of female flowers at a high temperature season. Overall, the NaCl salt was more effective on slowing down the stem elongation and had the lower incidence of leaf injury than the $CaCl_2$ salt.
Proceedings of the Korean Society of Toxicology Conference
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2002.11b
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pp.107-114
/
2002
Cholestatic liver injury results from the accumulation of toxic bile salts within the liver. The aim of the present study was to understand the mechanism of bile salts-induced hepatocellular apoptosis in bile duct-ligated (BDL) rats, using Western blot and immunohistochemical analysis.(omitted)
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