• Title/Summary/Keyword: SAMUL Play

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Investigation on the Perception Changes of the Korean Music through Developing A Teaching Method for Samul-nori (사물놀이 지도법 개발과 이를 통한 국악의 인식 변화 연구)

  • Lee, Ka-Won;Kim, Young-Won
    • The Journal of the Korea Contents Association
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    • v.12 no.3
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    • pp.114-122
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    • 2012
  • Traditional music should be succeeded and developed through the systematic education since music represents culture and history of one country. In Korea, even through the importance of Korean traditional music has been emphasized through the seven times of revision of national curriculum, music education has been still Western-oriented. This study aims to make students have experience Uttari Samul-nori and investigate the perception changes about the Korean traditional music. It is ultimately expected that students inherit the Korean traditional music and further recreate our own traditional culture. Samul-nori class was organized in the regular music curriculum twice a week, for 10 hours and various activities were tried during that time. 1st-year high school students participated in this research and they were allowed to play Samul instruments directly and play the basic rhythm suggested in the newly designed curriculum. Before and after the research, the questionnaires were sent to examine the attitude changes toward the Korean traditional music. The result of the questionnaire are as follows: First, Samul-nori activities affect positively the students' interest in the Korean traditional music and Samul-nori itself. Second, Danso(short bamboo flute) education which has been implemented most frequently during the Korean music education, is not satisfactory to the students. Third, students were satisfied with the new teaching method of Samul-nori and most students wanted to continue to take Samul-nori class. Last, students recognized the importance of Korean traditional music education after the research activities.

Protective Effects of Samul-tang on Oxidative Stress induced Death of H9c2 Cardioblast Cells (배양심근세포의 산화적 손상에 대한 사물탕의 방어효과)

  • Cho Kwon-Il;Jung Seung-Won;Jang Jae-Ho;Lee Dae-Yong;Park Sae-Wook;Lee In;Sin Sun-Ho;Moon Byung-Soon
    • The Journal of Korean Medicine
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    • v.26 no.1 s.61
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    • pp.174-186
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    • 2005
  • Objectives : The water extract of Samul-tang (SMT) has traditionally been used for treatment of ischemic heart and brain damage in oriental medicine. However, little is known about the mechanism by which the water extract of SMT rescues cells from these damages. Methods: This study was designed to investigate the protective mechanisms of SMT on oxidative stress-induced toxicity in H9c2 cardiomyoblast cells. Treatment with $H_2O_2$ markedly induced death of H9c2 cardiomyoblast cells in a dose-dependent manner. Results: The characteristics of H20z-induced death of H9c2 showed apparent apoptotic features such as DNA fragmentation and morphological change. However, SMT significantly reduced both H202-induced cell death and morphological change. The decrease of Bc-2 expression by High were inhibited by SMT. In addition, the increase of Bax expression was also inhibited by SMT. The cotreatment of SMT and $H_2O_2$ in H9c2 cells also induced the phosphorylation of ERK in a time-dependent manner. Moreover, PD98059, a specific inhibitor of ERK1/2 attenuated the protective effects of SMT on $H_2O_2-induced$ toxicity in H9c2 cardiomyoblast cells. These results suggest that both ERK1/2 signaling pathways play important roles in the protective effects of SMT on $H_2O_2-induced$ apoptotic death of H9c2 cells. Also, the expression profile of proteins in $H_2O_2$ cardiomyoblast cells were screened by using two-dimensional (2-D) gel electrophoresis. Among 300 spots resolved in 2-D gels, the comparison of control versus apoptosis cells revealed that signal intensity of 17 spots increased and 11 spots decreased. Conclusions: Taken together, this study suggests that the protectiw effects of the water extract of SMT against oxidative damages may be mediated by the modulation of Bc1-2 and Bax expression via the regulation of the ERK signaling pathway.

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