• Journal of the Korea Society of Computer and Information
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• v.13 no.6
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• pp.225-232
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• 2008

When a single source of multimedia contents is distributed to multiple reproduction devices, the audio and video contents require synchronous play for multi-channel stereo sound and lip-synchronization. The multimedia system in vehicle, especially, has researched to move to wireless environments from legacy wired environments. This paper proposes the advanced algorithm for providing synchronized services of real-time multimedia traffic in IEEE 802.11 WLANs [1]. For these, we implement the advanced IEEE 1588 Precision Time Protocol [2] and the environments for simulation. Also, we estimate and analysis performance of the algorithm, then we experiment and analysis after the porting of algorithm in wireless LAN devices (Linksys wrt-350n AP network device) to characterize timing synchronization accuracy.

• Journal of Institute of Control, Robotics and Systems
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• v.7 no.3
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• pp.198-208
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• 2001

In this paper, vibration suppression of an elastic beam fixed on a moving cart and carrying a fixed or moving mass is considered. A modified pulse sequence method with RIC(Robust Internal-loop Compensator) is proposed to suppress the single model residual vibration and to get accurate positioning of the beam-mass-cart system. The performance of the proposed input preshaping method is compared with that of the previous ones through simulations and experiments. Using the proposed method, it is able to suppress the initial vibration of the beam-mass-cart system carrying a concentrated mass. Accurate PTP(point-to-point) positioning of the moving mass without residual vibration is also obtained experimentally by modifying the proposed pulse sequence method. Finally, the proposed input preshaping method is applied successfully to the system to follow square trajectories of the moving mass without residual vibration.

• Biomedical Science Letters
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• v.18 no.1
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• pp.1-9
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• 2012

Differentially expressed genes by 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) were identified in order to evaluate them as dioxin-sensitive markers and crucial signaling molecules to understand dioxin-induced toxic mechanisms in human bronchial cells. Gene expression profiling was analyzed by cDNA microarray and ten genes were selected for further study. They were cytochrome P450, family 1, subfamily B, polypeptide 1 (CYP1B1), S100 calcium binding protein A8 (calgranulin A), S100 calcium binding protein A9 (calgranulin B), aldehyde dehydrogenase 1 family, member A3 (ALDH6) and peroxiredoxin 5 (PRDX5) in up-regulated group. Among them, CYP1B1 was used as a hallmark for dioxin and sharply increased by TCDD exposure. Down-regulated genes were IK cytokine, interferon-induced protein with tetratricopeptide repeats 1 (IFIT1), nuclease sensitive element binding protein 1 (NSEP1), protein tyrosine phosphatase type VI A, member 1 (PTP4A1), ras oncogene family 32 (RAB32). Although up-regulated 4 genes in microarray were coincided with northern hybridization, down-regulated 5 genes showed U-shaped expression pattern which is sharply decreased at lower doses and gradually increased at higher doses. These results introduce some of TCDD-responsive genes can be sensitive markers against TCDD exposure and used as signaling cues to understand toxicity initiated by TCDD inhalation in pulmonary tissues.

• Proceedings of the Korean Society for Agricultural Machinery Conference
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• 2002.02a
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• pp.471-478
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• 2002

초음파를 사과의 비파괴 품질판정에 이용하기 위한 기초연구로서 계측된 저장기간에 따른 사과의 초음파 특성과 본 연구에서 계측된 사과의 기계적 특성을 이용하여 초음파에 의한 사과의 탄성계수 및 생물체항복강도 예측모델을 개발하고자 하였으며, 결론은 다음과 같다. 1. UTM을 이용하여 사과의 기계적 특성치를 분석하여 생물체항복점, 생물체항복변형량, 생물체항복강도, 파괴점, 극한변형량, 극한강도 및 탄성계수 등을 구하였다. 2. 사과의 기본 물성, 초음파 특성과 기계적 특성값 들을 분석한 결과 사과의 질량, 체적, 시간영역의 진폭(PTP), 제3영역 에너지 스펙트럼 밀도함수가 기계적 특성 중 생물체항복강도, 탄성계수와 높은 상관성이 있는 것으로 나타났다. 3. 사과의 저장 기간, 질량, 체적, Peak-to-peak, 제3영역의 에너지값 등 5개의 독립변수를 가지는 다중선형회귀모형으로 사과의 탄성계수 및 생물체 항복강도 예측모형을 개발하였다.

• Transactions of the Korean Society of Automotive Engineers
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• v.13 no.4
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• pp.113-120
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• 2005

In order to investigate the characteristics of penatration and the effect of surface treatment in A15052-H34, Al5082-Hl31 and titanium alloy laminates which were treated by anodizing and PVD(Physical Vapor Desposition) method, ballistic tests were conducted. Thickness of surface membrane in A15052-H34, Al5082-Hl31, were $25{\mu}m$ and that of titanium $0.9{\mu}m$ respectively. Surface hardness test was conducted using micro Vicker's hardness tester. Resistance to penetration is determined by the protection ballistic limit(V50), a statistical velocity with $50\%$ probability for complete penetration. Fracture behaviors and ballistic tolerance, described by penetration modes, are observed from the results of V50 test and Projectile Through Plate(PTP) test at velocities greater than protection ballistic limit, respectively. Present experimental results derived from this research help to optimize laminate impact behavior by varing the laminate thickness and surface treated materials.

• Korean Journal of Microbiology
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• v.34 no.4
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• pp.243-247
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• 1998

Comparative analyses of intracellular biopterin contents and its biosynthetic enzymes were performed in Allomyces macrogynus. Biopterin content in fresh weight was 14-fold higher in mycelium than in zoospore. Enzyme activities of GTP cyclohydrolase I and 6-pyruvoyltetrahydropterin synthase in ammonium sulfate fractions were approximately 2-fold higher in mycelium. On the other hand, sepiapterin reductase (SR) activity was 10 fold higher in zoospore. Northern blot assay also demonstrated that SR transcript was abundant in zoospore. These results suggest a possible involvement of tetrahydrobiopterin in cellular differentiation of Allomyces macrogyllus as well as provide an experimental basis to elucidate the physiological function of SR in this organism.

• KSII Transactions on Internet and Information Systems (TIIS)
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• v.15 no.7
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• pp.2321-2338
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• 2021

Compared with vehicle trajectories, pedestrian trajectories have stronger degrees of freedom and complexity, which poses a higher challenge to trajectory prediction tasks. This paper designs a mode to divide the trajectory of pedestrians at a traffic intersection, which converts the trajectory regression problem into a trajectory classification problem. This paper builds a deep model for pedestrian trajectory prediction at intersections for the task of pedestrian short-term trajectory prediction. The model calculates the spatial correlation and temporal dependence of the trajectory. More importantly, it captures the interactive features among pedestrians through the Attention mechanism. In order to improve the training speed, the model is composed of pure convolutional networks. This design overcomes the single-step calculation mode of the traditional recurrent neural network. The experiment uses Vulnerable Road Users trajectory dataset for related modeling and evaluation work. Compared with the existing models of pedestrian trajectory prediction, the model proposed in this paper has advantages in terms of evaluation indicators, training speed and the number of model parameters.

• The Korean Journal of Physiology and Pharmacology
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• v.5 no.2
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• pp.139-146
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• 2001

L-type $Ca^{2+}$ channels play an important role in regulating cytosolic $Ca^{2+}$ and thereby regulating hormone secretions in neuroendocrine cells. Since hormone secretions are also regulated by various kinds of protein kinases, we investigated the role of some kinase activators and inhibitors in the regulation of the L-type $Ca^{2+}$ channel currents in rat pituitary $GH_3$ cells using the patch-clamp technique. Phorbol 12,13-dibutyrate (PDBu), a protein kinase C (PKC) activator, and vanadate, a protein tyrosine phosphatase (PTP) inhibitor, increased the $Ba^{2+}$ current through the L-type $Ca^{2+}$ channels. In contrast, bisindolylmaleimide I (BIM I), a PKC inhibitor, and genistein, a protein tyrosine kinase (PTK) inhibitor, suppressed the $Ba^{2+}$ currents. Forskolin, an adenylate cyclase activator, and isobutyl methylxanthine (IBMX), a non-specific phosphodiesterase inhibitor, reduced $Ba^{2+}$ currents. The above results show that the L-type $Ca^{2+}$ channels are activated by PKC and PTK, and inhibited by elevation of cyclic nucleotides such as cAMP. From these results, it is suggested that the regulation of hormone secretion by various kinase activity in $GH_3$ cells may be attributable, at least in part, to their effect on L-type $Ca^{2+}$ channels.

• Analytical Science and Technology
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• v.33 no.5
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• pp.215-223
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• 2020

The objectives of the present study were to assess the oxidative stability of South Korean n-3 (omega-3 fatty acid) supplements carried out from 2018 to 2019 and evaluate the influence of product characteristics on oxidative safety. A total of 76 n-3 supplements were analysed for oxidation safety by four markers, including acid value (AV), primary oxidation (peroxide value, PV), secondary oxidation (p-anisidine value, pAV) and total oxidation value (TOTOX). Among the supplements tested, 5.3 %, 55.3 %, 28.9 % and 46.1 % exceeded the international voluntary recommended levels for AV, PV, pAV and TOTOX, respectively. Purity (%) of products, remainder of expiration date (suggested shelf life), package in press through package (PTP) and products with additives had statistically significant differences oxidation assessment levels (p < 0.05). In addition, n-3 group found in Algae oil had significantly lower AV levels than the group that did not, and product with Alaska pollack oil, had significantly higher pAV levels than without group (p < 0.05). The high oxidation status of South Korean n-3 products in the present study could not be considered a public health problem right now. However, the levels of oxidation may affect a lot the efficacy and safety of using n-3 supplements. Thus, current oxidation safety limits should be reestablished by regulatory bodies to ensure the safety and efficacy of n-3 supplements, so that the standards could be applied to the products available to consumers.

• Bulletin of the Korean Chemical Society
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• v.29 no.2
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• pp.342-346
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• 2008

Aurintricarboxylic acid (ATA) prevents apoptosis in a wide range of cell types, including PC12 cells. ATA is known to increase the phosphorylation level of IGF-1 receptor (IGF-1R) and downstream signaling proteins. ATA can translocate across the plasma membrane of PC12 cells and inhibit protein tyrosine phosphatases (PTPs) and, therefore, it is not clear whether ATA exerted its antiapoptotic effect through activation of IGF-1R or by inhibition of cytosolic PTPs. When PC12 cells, deprived of serum, were treated with Fab fragment of anti-IGF-1R antibody to prevent the binding of ATA to the extracellular domain of IGF-1R, ATA was found to penetrate into the cytosolic space of the cells. Under these conditions, the survival-promoting effects of ATA were abolished, and the increase of phosphorylation and characteristic cleavage of IGF-1R were not observed. These results indicate that the antiapoptotic effect of ATA in PC12 cells is due to the binding of ATA to the extracellular domain of IGF-1R and subsequent activation of the IGF-1R, not inhibition of cytosolic PTP(s).