Journal of Korean Society for Atmospheric Environment
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v.18
no.4
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pp.285-295
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2002
In this study, several types of gravimetric methods (such as high, medium, low, and ultra low volume sampling methods) were applied to determine suspended particulate matter concentrations in both ambient and indoor environments. Comparative evaluations were undertaken with SPM data obtained using a variety of samplers (TSP, PM10, and PM4.0) at different sampling flow rates. Correlation coefficients between TSP and PM10 concentrations measured at different flow rates fell in the range of 0.73∼0.94 (n=40). In addition, correlation coefficients for PM concentrations measured by different TSP samplers were in the range of 0.90∼0.95 (n=36 or n=38), while 0.77∼0.91 (n=38) for PM10 samplers. Correlation analysis was also conducted on indoor monitoring data that were measured using ultra-low-volume samplers at both different or identical flow rates. The correlation coefficients were in the range of 0.98∼0.99 (n=38) between TSP and TSP and 0.92∼0.94 (n=38) between TSP and PM10. The mean ratio for high volume PM10 to TSP concentration that was monitored at identical flow rates in the ambient air appeared to be 0.72. The mean ratios of PM10 to TSP and PM4.0 to TSP observed with identical flow rates at indoor environments were 0.47 and 0.40. The results of this study may provide empirical information concerning the compatability of aerosol data obtained by gravimetric sampling methods at different flow rates.
Journal of the Korean Society of Surveying, Geodesy, Photogrammetry and Cartography
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v.37
no.6
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pp.417-426
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2019
Conventional visualization of PM (Particulate Matter)10 flows applies superimposition of concentration distribution maps and wind field maps. This method is efficient for small scale maps where only macro flow trends are of interest. However, in the case of urban areas, local flows are difficult to model at micro level using wind fields, and therefore different methods of flow extraction is deemed necessary. In this study, flow information is extracted and visualized directly from the PM10 density data by using the gravity model. This method has the advantage that additional information such as wind field is not necessary for estimating the intensity and direction of PM10 flow. The extracted spatio-temporal flow patterns of PM10 are analyzed with relation to traffic information.
Purpose: In this single repeated measures study, an examination was done on the effects of dialysate flow rate on dialysis adequacy and fatigue in patients receiving hemodialysis. Methods: This study was a prospective single center study in which repeated measures analysis of variance were used to compare Kt/V urea (Kt/V) and urea reduction ratio (URR) as dialysis adequacy measures and level of fatigue at different dialysate flow rates: twice as fast as the participant's own blood flow, 500 mL/min, and 700 mL/min. Thirty-seven hemodialysis patients received all three dialysate flow rates using counterbalancing. Results: The Kt/V ($M{\pm}SD$) was $1.40{\pm}0.25$ at twice the blood flow rate, $1.41{\pm}0.23$ at 500 mL/min, and $1.46{\pm}0.24$ at 700 mL/min. The URR ($M{\pm}SD$) was $68.20{\pm}5.90$ at twice the blood flow rate, $68.67{\pm}5.22$ at 500 mL/min, and $70.11{\pm}5.13$ at 700 mL/min. When dialysate flow rate was increased from twice the blood flow rate to 700 mL/min and from 500 mL/min to 700 mL/ min, Kt/V and URR showed relative gains. There was no difference in fatigue according to dialysate flow rate. Conclusion: Increasing the dialy-sate flow rate to 700 mL/min is associated with a significant increase in dialysis adequacy. Hemodialysis with a dialysate flow rate of 700 mL/min should be considered in selected patients not achieving adequacy despite extended treatment times and optimized blood flow rate.
Water vapor has received worldwide large attention due to its broad technological implications ranged from resource production and environmental remediation. Especially, one of the typical areas where the water vapor is important is the removal of PM (particulate matter) which causes a critical hazard to human health. However, most vapor-based PM removal methods are limited in removing PM2.5 by using relatively large water droplets and consume large energy. Here, we propose a superhydrophilic thermally-insulated macroporous membrane to generate steam flow. The water vapor directly captures PM with steam flow and hygroscopic characteristic of PM. The steam, the cluster of water vapor, from the membrane gives rise to high removal efficiencies compared to those of the control case without light illumination. To reveal PM removal mechanism, the steam flow and PM were quantitatively analyzed using PIV measurement. The proposed steam generator could be utilized as an economical and ecofriendly platform for effective PM removal at a fairly low cost in a sustainable, energy-free, and harmless-to-human manner.
Particulate matter (PM) is designated as a group 1 carcinogen by the International Agency for Research on Cancer (IARC) of the World Health Organization (WHO). In South Korea, the health threat caused by PM is the most serious level internationally. Therefore, in order to solve the urban PM problem, it is important to develop the technology that can control PM efficiently. In this study, CFD(Computational Fluid Dynamics) simulation was performed for PM pre-filter (type 1-3 with different PM collecting room) to develop a high-efficiency PM collecting device. The complex flow field and the local flow phenomenon inside the PM collecting device were understood with CFD simulation by changing the shape and size of the pre-filter. The PM removal performance can be described with flow rate through the device and PM removal efficiency. The type-1 pre-filter with 5x5 size collecting room was confirmed to have the highest efficiency. Based on the analysis results, the optimal type of pre-filter could be developed and it would be applied as an element technology included in the PM collecting device.
Ginsenosides are one of the most well-known traditional herbal medicines frequently used for the treatment of cardiovascular symptoms in korea. The anti-ischemic effects of the mixture of ginsenoside $Rg_3$, and CK on ischemia-induced isolated rat heart were investigated through analyses of changes in hemodynamics ; blood pressure, aortic flow, coronary flow, and cardiac output. The subjects in this study were divided into four groups: normal control, the mixture of ginsenoside $Rg_3$ and CK, an ischemia-induced group without any treatment, and an ischemia-induced group treated with the mixture of ginsenoside $Rg_3$ and CK. There were no significant differences in perfusion pressure, aortic flow, coronary flow and cardiac output between them before ischemia was induced. The supply of oxygen and buffer was stopped for five minutes to induce ischemia in isolated rat hearts, and the mixture of ginsenoside $Rg_3$ and CK was administered during ischemia induction. Treatments of the mixture of ginsenoside $Rg_3$ and CK significantly prevented decreases in perfusion pressure, aortic flow, coronary flow, and cardiac output under ischemic conditions. In addition, hemodynamics (except heart rate) of the group treated with the mixture of ginsenoside $Rg_3$ and CK significantly recovered 60 minutes after reperfusion compared to the control group (mixture+ischemia vs ischemia - average perfusion pressure: 74.4${\pm}$2.97% vs. 85.1${\pm}$3.01%, average aortic flow volume: 49.11${\pm}$2.72% vs. 59.97${\pm}$2.93%, average coronary flow volume: 58.50${\pm}$2.81% vs. 72.72${\pm}$2.99%, and average cardiac output: 52.47${\pm}$2.78% vs. 63.11${\pm}$2.76%, p<0.01, respectively). These results suggest that treatment of the mixture of ginsenoside $Rg_3$ and CK has distinct anti-ischemic effects in ex vivo model of ischemia-induced rat heart.
In 20 normal cases and 39 pulmonary tuberculosis cases, regional pulmonary arterial blood flow measurements and lung perfusion scans by $^{131}I$-Macroaggregated albumin, lung inhalation scans by colloidal $^{198}Au$ and spirometries by respirometer were done at the Radiological Research Institute. The measured lung function tests were compared and the results were as the following: 1. The normal distribution of pulmonary blood flow was found to be $54.5{\pm}2.82%$ to the right lung and $45.5{\pm}2.39%$ to the left lung. The difference between the right and left pulmonary arterial blood flow was significant statistically (p<0.01). In the minimal pulmonary tuberculosis, the average distribution of pulmonary arterial blood flow was found to be $52.5{\pm}5.3%$ to the right lung and $47.5{\pm}1.0%$ to the left lung when the tuberculous lesion was in the right lung, and $56.2{\pm}4.4%$ to the right lung and $43.8{\pm}3.1%$ to the left lung when the tuberculous lesion was in the left lung. The difference of pulmonary arterial blood flow between the right and left lung was statistically not significant compared with the normal distribution. In the moderately advanced pulmonary tuberculosis, the average distripution of pulmonary arterial blood flow was found to be $26.9{\pm}13.9%$ to the right lung and $73.1{\pm}13.9%$ to the left lung when the tuberculous lesion was more severe in the right lung, and $79.6{\pm}12.8%$ to the right lung and $20.4{\pm}13.0%$ to the left lung when the tuberculous lesion was more severe in the left lung. These were found to be highly significant statistically compared with the normal distribution of pulmonary arterial blood flow (p<0.01). When both lungs were evenly involved, the average distribution of pulmonary arterial blood flow was found to be $49.5{\pm}8.01%$ to the right lung and $50.5{\pm}8.01%$ to the left lung. In the far advanced pulmonary tuberculosis, the average distribution of pulmonary arterial blood flow was found to be $18.5{\pm}11.6%$ to the right lung and $81.5{\pm}9.9%$ to the left lung when the tuberculous lesion was more severe in the right lung, and $78.2{\pm}8.9%$ to the right lung and $21.8{\pm}10.5%$ to the left lung when the tuberculous lesion was more severe in the left lung. These were found to be highly significant statistically compared with the normal distribution of pulmonary arterial blood flow (p<0.01). When both lungs were evenly involved the average distribution of pulmonary arterial blood flow was found to be $56.0{\pm}3.6%$ to the right lung and $44.0{\pm}3.2%$ to the left lung. 2. Lung perfusion scan by $^{131}I$-MAA in patients with pulmonary tuberculosis was as follows: a) In the pretreated minimal pulmonary tuberculosis, the decreased area of pulmonary arterial blood flow was corresponding to the chest roentgenogram, but the decrease of pulmonary arterial blood flow was more extensive than had been expected from the chest roentgenogram in the apparently healed minimal pulmonary tuberculosis. b) In the pretreated moderately advanced pulmonary tuberculosis, the decrease of pulmonary arterial blood flow to the diseased area was corresponding to the chest roentgenogram, but the decrease of pulmonary arterial blood flow was more extensive in the treated moderately advanced pulmonary tuberculosis as in the treated minimal pulmonary tuberculosis. c) Pulmonary arterial blood flow in the patients with far advanced pulmonary tuberculosis both before and after chemotherapy were almost similar to the chest roentgenogram. Especially the decrease of pulmonary arterial blood flow to the cavity was usually greater than had been expected from the chest roentgenogram. 3. Lung inhalation scan by colloidal $^{198}Au$ in patients with pulmonary tuberculosis was as follows: a) In the minimal pulmonary tuberculosis, lung inhalation scan showed almost similar decrease of radioactivity corresponding to the chest roentgenogram. b) In the moderately advanced pulmonary tuberculosis the decrease of radioactivity in the diseased area was partly corresponding to the chest roentgenogram in one hand and on the other hand the radioactivity was found to be normally distributed in stead of tuberculous lesion in the chest roentgenogram. c) In the far advanced pulmonary tuberculosis, lung inhalation scan showed almost similar decrease of radioactivity corresponding to the chest roentgenogram as in the minimal pulmonary tuberculosis. 4. From all these results, it was found that the characteristic finding in pulmonary tuberculosis was a decrease in pulmonary arterial blood flow to the diseased area and in general decrease of pulmonary arterial blood flow to the diseased area was more extensive than had been expected from the chest roentgenogram, especially in the treated group. Lung inhalation scan showed almost similar distribution of radioactivity corresponding to the chest roentgenogram in minimal and far advanced pulmonary tuberculosis, but there was a variability in the moderately advanced pulmonary tuberculosis. The measured values obtained from spirometry were parallel to the tuberculous lesion in chest roentgenogram.
Journal of the korean academy of Pediatric Dentistry
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v.39
no.4
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pp.383-388
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2012
The aim of this study was to compare the compressive strength and the surface microhardness of Beautifil flow (Shofu, Kyoto, Japan) with $Filtek^{TM}$ Z350, Z350XT (3M ESPE, USA). Fifteen specimens from each material were fabricated for testing. Compressive strength was measured by using a universal testing machine at a crosshead speed of 1 mm/min. Surface microhardness values were measured by using Vickers hardness tester under 4.9 N load and 10 seconds dwelling time. The compressive strength of Group 2 $Filtek^{TM}$ Z350XT shows the highest value as $218.7{\pm}18.4$ MPa and Group 1 $Filtek^{TM}$ Z350 was $205.5{\pm}27.1$ MPa. Group 3 Beautifil flow F00 was $176.5{\pm}30.3$ MPa, and Group 4 Beautifil flow F10 was $173.4{\pm}26.2$ MPa. The compressive strength of Group 2 is higher than Group 3 and 4 (p < 0.05). The surface microhardness of Group 2 $Filtek^{TM}$ Z350XT shows the highest value as $39.1{\pm}2.1$ and Group 4 Beautifil flow F10 was $27.9{\pm}1.8$. And Group 3 Beautifil flow F00 was $23.1{\pm}1.1$, Group 1 $Filtek^{TM}$ Z350 was $20.4{\pm}0.9$. There was a statistical significant difference in surface microhardness between all groups (p < 0.05). In conclusion, the compressive strength of giomer was below the level of flowable composite resin. However, the surface microhardness of giomer is comparable to that of flowable composite resin. Giomer would be the good alternative to composite resin, if there is improvement of the compressive strength of giomer.
The purpose of this study was to investigate the functional involvement of sympathetic nerve in the control of the microcirculation in the dental pulp with the aim of elucidation of the involvement of neuropeptides and sympathetic nerve in neurogenic inflammation. Experiments were done on the 7 cats anesthetised with sodium pentobarbital, and sympathetic nerve to the' dental pulp was stimulated electrically (10 Hz, 4 V, 1.5 ms, 3.5 mins). Ana-adrenoceptor antagonist phentolamine and a neuropeptide Y antagonist D-myo-inositol-1,2,6-trisphosphate (PP56) were injected close intra-arterially into the dental pulp without changing the systemic blood pressure. The probe of laser Doppler flowmeter was placed on the buccal surface of ipsilateral canine teeth to the stimulation, and pulpal blood flow was measured. Stimulation of the sympathetic nerve decreased pulpal blood flow by $55.24{\pm}7.74\;%$ (mean${\pm}$SEM, n = 13). Stimulation of the sympathetic nerve following the injection of the ${\alpha}$-adrenoceptor antagonist phentolamine ($0.1{\mu}g$/kg) caused decrease of pulpal blood flow by $14.35{\pm}3.43%$ (mean${\pm}$SEM, n=5). Phentolamine attenuated the sympathetic nerve-induced pulpal blood flow decrease by $74.02{\pm}9.32%$ (mean${\pm}$SEM) Stimulation of the sympathetic nerve following the injection of the neuropeptide Y antagonist PP56 (2.3 mg/kg) caused decrease of pulpal blood flow by $30.64{\pm}7.92%$ (mean${\pm}$SEM, n=6). PP56 attenuated the sympathetic nerve-induced pulpal blood flow decrease by $44.37{\pm}11.01%$ (mean${\pm}$SEM). These data provide evidences of the co-contribution of nerepinephrine and neuropeptide Y on the sympathetic nerve-induced vasoconstriction in the feline dental pulp. In addition, they show functional evidences that sympathetic nerve plays an active role in controlling the microcirculation of the dental pulp.
To evaluate the acute effects of fine particles on pulmonary function, a longitudinal study was conducted. This study was carried out for the schoolchildren (3rd and 6th grades) living in Beijing, China. Each child was provided with a mini-Wright peak flow meter and a preformatted health symptom diary for 40 days, and was trained on their proper use. Participants were instructed to perform the peak flow test three times in standing position, three times a day (9 am, 12 pm, and 8 pm), and to record all the readings along with the symptoms (cold, cough, and asthmatic symptoms) experienced on that day. Daily measurement of fine particles (PM$_{10}$ and PM$_{2.5}$) was obtained in the comer of the playground of the participating elementary school for the same period of this longitudinal study. The relationship between daily peak expiratory flow rate (PEFR) and fine particle levels was analyzed using a mixed linear regression models including gender, height, the presence of respiratory symptoms, and daily average temperature and relative humidity as extraneous variables. The total number of students participating in this longitudinal study was 87. The range of daily measured PEFR was 253-501$\ell$/min. In general, the PEFR measured in the morning was lower than the PEFR measured in the evening (or afternoon) on the same day. The daily mean concentrations of PM$_{10}$ and PM$_{2.5}$ over the study period were 180.2$\mu\textrm{g}$/㎥ and 103.2$\mu\textrm{g}$/㎥, respectively. The IQR (inter-quartile range) of PM$_{10}$ and PM$_{2.5}$ were 91.8$\mu\textrm{g}$/㎥ and 58.0$\mu\textrm{g}$/㎥. During the study period, the national ambient air quality standard of 150$\mu\textrm{g}$/㎥ (for PM$_{10}$) was exceeded in 23 days (57.5%). The analysis showed that an increase of 1$\mu\textrm{g}$/㎥ of PM$_{10}$ corresponded to 0.59$\mu\textrm{g}$/㎥ increment of PM$_{2.5}$. Daily mean PEFR was regressed with the 24-hour average PM$_{10}$ (or PM$_{2.5}$) levels, weather information such as air temperature and relative humidity, and individual characteristics including gender, height, and respiratory symptoms. The analysis showed that the increase of fine particle concentrations was negatively associated with the variability in PEFR. The IQR increments of PM$_{10}$ or PM$_{2.5}$ (at 1-day time lag) were also shown to be related with 1.54 $\ell$/min (95% Confidence intervals: 0.94-2.14) and 1.56$\ell$/min (95% CI: 0.95-2.16) decline in PEFR.R.ine in PEFR.ine in PEFR.
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