• Title/Summary/Keyword: Neurogenic thoracic outlet syndrome

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A Case of True Neurogenic Thoracic Outlet Syndrome by Cervical Rib (경늑골에 의한 신경인성 가슴문증후군 1예)

  • Lee, Da-In;Kim, Sun-Young;Suh, Chung-Kyu
    • Annals of Clinical Neurophysiology
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    • v.11 no.1
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    • pp.28-32
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    • 2009
  • True neurogenic thoracic outlet syndrome (NTOS) is an extremely rare condition, probably occurring with an incidence of one per million. It is often misdiagnosed as carpal tunnel syndrome or benign focal amyotrophy, and careful clinical examination, electrodiagnostic studies, and imaging studies are essential for the correct diagnosis. Here, we report a 23-year-old woman with slowly progressive weakness and atrophy of abductor pollicis brevis who were found to have NTOS caused by cervical rib.

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Surgery for Entrapments of the Thoracic Outlet (흉곽 출구 포착성 신경 병증의 외과적 치료)

  • Chung, Hwan-Yung
    • Archives of Reconstructive Microsurgery
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    • v.8 no.1
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    • pp.1-9
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    • 1999
  • Surgical treatment was performed on the 39 cases out of 76 cases of entrapments of the thoracic outlet. The remaining 36 cases of entrapments were treated by conservatively. The operated cases were categorized as follows. They were 34 cases of scalenus anticus syndrome, 1 of cervical rib syndrome, 2 of costoclavicular syndrome, and 2 of hyperabduction syndrome. 1. Scalenus anticus syndrome : Anterior scalenotomy was performed by simple sectioning of the attachment to the first rib. 2. Cervical rib syndrome : Complete decompressive resection of cervical rib sometimes required both anterior and posterior approaches to avoid over-retraction of the brachial plexus. 3. Costoclavicular syndrome : Partial decompressive claviculectomy was undergone instead of conventional total claviculectomy. 4. Hyperabduction syndrome : The resection of coracoid process was performed as well as conventional tenotomy of pectoralis minor muscle to insure free up-and-down moving of neurovascular bundle at the time of hyperabduction. Every diagnostic maneuver was tested at the time of operation to observe whether or not neurovascular decompression including restoration of radial pulse was sufficient. Despite of the postoperative vascular restoration was inmediate, neurogenic symptoms were improved slowly. Because this entity is essentially chronic nerve injnry, its recovery needed a couple of months or several. Although improvement was slow, ultimate results were definite. Complication was not observed.

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