• Journal of Radiopharmaceuticals and Molecular Probes
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• v.7 no.1
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• pp.1-2
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• 2021

• Journal of Radiopharmaceuticals and Molecular Probes
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• v.6 no.2
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• pp.59-60
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• 2020

• Journal of Radiopharmaceuticals and Molecular Probes
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• v.6 no.1
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• pp.1-2
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• 2020

• Clinical and Experimental Reproductive Medicine
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• v.44 no.4
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• pp.159-170
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• 2017

The development of biomarkers of reproductive medicine is still in its infancy because many black boxes are still present in reproductive medicine. Novel approaches to human infertility diagnostics and treatment must be developed because reproductive medicine has lagged behind in the implementation of biomarkers in clinical medicine. Despite the dearth of the available literature, the current rapid pace of publications suggests that this gap will soon be filled therefore; this review is a $pr\acute{e}cis$ of the research that has been done so far and will provide a basis for the development of biomarkers in reproductive medicine.

• Tuberculosis and Respiratory Diseases
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• v.83 no.2
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• pp.107-115
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• 2020

Aging is often viewed as a progressive decline in fitness due to cumulative deleterious alterations of biological functions in the living system. Recently, our understanding of the molecular mechanisms underlying aging biology has significantly advanced. Interestingly, many of the pivotal molecular features of aging biology are also found to contribute to the pathogenesis of chronic lung disorders such as chronic obstructive pulmonary disease and idiopathic pulmonary fibrosis, for which advanced age is the most crucial risk factor. Thus, an enhanced understanding of how molecular features of aging biology are intertwined with the pathobiology of these aging-related lung disorders has paramount significance and may provide an opportunity for the development of novel therapeutics for these major unmet medical needs. To serve the purpose of integrating molecular understanding of aging biology with pulmonary medicine, in this review, recent findings obtained from the studies of aging-associated lung disorders are summarized and interpreted through the perspective of molecular biology of aging.

• Asian Pacific Journal of Cancer Prevention
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• v.15 no.3
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• pp.1501-1501
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• 2014

• Proceedings of the Microbiological Society of Korea Conference
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• 2003.05a
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• pp.97-99
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• 2003

• Proceedings of the Korean Society of Life Science Conference
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• 2002.09a
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• pp.89-89
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• 2002

• Proceedings of the Zoological Society Korea Conference
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• 1999.10b
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• pp.36-36
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• 1999

No Abstract, See Full Text

• BMB Reports
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• v.56 no.7
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• pp.404-409
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• 2023

This study investigates the relationship between cancer cachexia and the gut microbiota, focusing on the influence of cancer on microbial composition. Lewis lung cancer cell allografts were used to induce cachexia in mice, and body and muscle weight changes were monitored. Fecal samples were collected for targeted metabolomic analysis for short chain fatty acids and microbiome analysis. The cachexia group exhibited lower alpha diversity and distinct beta diversity in gut microbiota, compared to the control group. Differential abundance analysis revealed higher Bifidobacterium and Romboutsia, but lower Streptococcus abundance in the cachexia group. Additionally, lower proportions of acetate and butyrate were observed in the cachexia group. The study observed that the impact of cancer cachexia on gut microbiota and their generated metabolites was significant, indicating a host-to-gut microbiota axis.