• 대한전자공학회논문지SD
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• 제48권4호
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• pp.39-50
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• 2011

본 논문에서는 패널 내부 인터페이스의 하나인 2세대 AiPi+의 클록-데이터 복원 회로(Clock & Data Recovery)를 제안하였다. 제안하는 클록-데이터 복원 회로의 속도는 기존 AiPi+ 보다 빠른 1.25 Gbps 로 향상되었으며 다중 위상 클록을 생성하기 위하여 Delay-Locked Loop(DLL)를 사용하였다. 본 논문에서는 패널 내부 인터페이스의 저전력, 작은 면적의 이슈를 만족하는 클록-데이터 복원 회로를 설계하였다. 매우 간단한 방법으로 자동적으로 Harmonic-locking 문제를 해결할 수 있는 주파수 검출기 구조를 제안하여 기존 주파수 검출기(Frequency Detector)의 복잡도, 전류 소모, 그리고 외부 인가에 따른 문제를 개선하였으며, 전압 제어 지연 라인(Voltage Controlled Delay Line) 에서 상승/하강 시간 차이에 따른 에지의 사라짐 현상을 막기 위해서 펄스 폭의 최대치를 제한하는 펄스 폭 오류 보정 방법을 사용하였다. 제안하는 클록-데이터 복원 회로는 CMOS 0.18 ${\mu}m$ 공정으로 제작되었으며 면적은 $660\;{\mu}m\;{\times}\;250\;{\mu}m$이고, 공급 전압은 1.8 V이다. Peak-to-Peak 지터는 15 ps, 입력 버퍼, 이퀄라이저, 병렬화기를 제외한 클록-데이터 복원 회로의 소모 전력은 5.94 mW 이다.

• 한국진공학회:학술대회논문집
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• 한국진공학회 2009년도 제38회 동계학술대회 초록집
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• pp.362-362
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• 2010

Today, vast attention has been paid to periodic arrays of nanostructures due to their potential for applications such as memory with huge storage density. Such application requires large-scale fabrication of well ordered nano-sized structures. One of the most widely used methods for the ordered nanostructures is lithography. This top-down process, however, has the limit to reduce size. Here the promising alternative is the self-organization of ordered nano-sized structures such as large scale 2d carbon-induced reconstructions on W(110). In the present study, we report on the first well-resolved atomic resolution STM studies of the well-known R($15{\times}3$) and R($15{\times}12$) carbon induced reconstruction of the W(110). From the atomic image of R($15{\times}3$) for different values of tunneling gap resistance, we can tell there are no missing atoms in unit cells of R($15{\times}3$) and some atomic displacements are substantial from the clean W(110), even though not all the imaged position of atoms correspond to tungsten, but may include those of carbon. We are considering two cases; First case is related to lattice deformation, or top layer of W(110) is deformed in the process of relief of strain caused by random inserting of carbon atoms possibly in the interstitial position. In the second case, R($15{\times}3$) unit cell results from a coincidence lattice between clean W(110) substrate and tungsten carbide overlayer which has rectangular atomic arrangement and giving R($15{\times}3$) coincidence lattice. beta-W2C showing rectangular unit cell should be a candidate. Further, we report on new reconstructions. Unlike the well-known R($15{\times}12$) consisting of two parts, two inner structures between two "Backbone" structures. The new reconstruction, which we found for the first time, contains more parts between the "Backbone"s. Sometimes we can observe the reconstruction consists of only inner parts without "Backbone" parts. Thus, the observed reconstruction can be built by constructing of two types of "Lego"-like block. Moreover, the rectangle shape of "Backbone" transform to parallelogram-like shape over time, the so-called wavy-R($15{\times}12$). Adsorption of hydrogen can be the reason for this transformation.

• Pediatric Gastroenterology, Hepatology & Nutrition
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• 제24권2호
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• pp.173-186
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• 2021

Purpose: Biliary atresia (BA) is a disease that manifests as jaundice after birth and leads to progressive destruction of the ductal system in the liver. The aim of this study was to investigate histopathological changes and immunohistochemically examine the expression of glial cell line-derived neurotrophic factor (GDNF), synaptophysin, and S-100 protein in the gallbladder of BA patients. Methods: The study included a BA group of 29 patients and a control group of 41 children with cholecystectomy. Gallbladder tissue removed during surgery was obtained and examined immunohistochemically and histopathologically. Tissue samples of both groups were immunohistochemically assessed in terms of GDNF, S-100 protein, and synaptophysin expression. Expression was classified as present or absent. Inflammatory activity assessment with hematoxylin and eosin staining and fibrosis assessment with Masson's trichrome staining were performed for tissue sample sections of both groups. Results: Ganglion cells were not present in gallbladder tissue samples of the BA group. Immunohistochemically, GDNF, synaptophysin, and S-100 expression was not detected in the BA group. Histopathological examination revealed more frequent fibrosis and slightly higher inflammatory activity in the BA than in the control group. Conclusion: We speculate that GDNF expression will no longer continue in this region, when the damage caused by inflammation of the extrahepatic bile ducts reaches a critical threshold. The study's findings may represent a missing link in the chain of events forming the etiology of BA and may be helpful in its diagnosis.