• 제목/요약/키워드: ERX

검색결과 4건 처리시간 0.018초

ERX : 개체 관계 모델로부터 XML 스키마 생성 도구 (ERX : A Generation Tool of XML Schema based on Entity-Relationship Model)

  • 김영웅
    • 한국인터넷방송통신학회논문지
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    • 제13권2호
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    • pp.149-155
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    • 2013
  • 오늘날 대표적인 데이터베이스 설계 도구로 개체 관계 모델을 사용하고 있으며, 데이터를 표현하고 교환하는 표준 언어로 XML을 사용하고 있다. 그러나 많은 개체 관계 모델 제품들은 각각 서로 다른 표현형식을 사용하기 때문에 이들 제품들 사이에 호환성에 어려운 점이 있으며, XML은 언어가 갖는 복잡성으로 인해 XML을 이용하여 현실세계를 직접 설계하기에는 어려운 점이 있다. 본 논문은 이질적인 제품들 사이의 호환성을 제공하기 위해 개체 관계 모델을 XML 스키마로 변환하는 도구인 ERX(Entity-Relationship model to Xml)를 제안한다. ERX는 입력으로 개체 관계 다이어그램을 받아 이를 변환 규칙을 통해 XML 스키마를 출력한다. 변환 규칙에는 개체집합, 관계집합, 매핑 대응수(mapping cardinality), 일반화(generalization) 개념 등을 포함한다.

Real-time implementation of distributed beamforming for simultaneous wireless information and power transfer in interference channels

  • Hong, Yong-Gi;Hwang, SeongJun;Seo, Jiho;Lee, Jonghyeok;Park, Jaehyun
    • ETRI Journal
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    • 제43권3호
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    • pp.389-399
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    • 2021
  • In this paper, we propose one-bit feedback-based distributed beamforming (DBF) techniques for simultaneous wireless information and power transfer in interference channels where the information transfer and power transfer networks coexist in the same frequency spectrum band. In a power transfer network, multiple distributed energy transmission nodes transmit their energy signals to a single energy receiving node capable of harvesting wireless radio frequency energy. Here, by considering the Internet-of-Things sensor network, the energy harvesting/information decoding receivers (ERx/IRx) can report their status (which may include the received signal strength, interference, and channel state information) through one-bit feedback channels. To maximize the amount of energy transferred to the ERx and simultaneously minimize the interference to the IRx, we developed a DBF technique based on one-bit feedback from the ERx/IRx without sharing the information among distributed transmit nodes. Finally, the proposed DBF algorithm in the interference channel is verified through the simulations and also implemented in real time by using GNU radio and universal software radio peripheral.

Regulation of SPIN90 by Cell Adhesion and ERK Activation

  • Kim Sung Hyun;Kim Dae Joong;Song Woo Keun
    • 한국미생물학회:학술대회논문집
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    • 한국미생물학회 2004년도 International Meeting of the Microbiological Society of Korea
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    • pp.141-146
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    • 2004
  • SPIN90 was identified to farm molecular complex with $\betaPIX$, WASP and Nck. This complex shows that SPIN90 interacts with Nck in a manner dependent upon cell adhesion to extracellular matrix, but $SPIN90{\cdot}{\beta}PIX{\cdot}WASP$ complex was stable even in suspended cells. This suggests that SPIN90 serves as an adaptor molecule to recruit other proteins to Nck at focal adhesions. SPIN90 was phosphorylated by ERK1, which was, itself, activated by cell adhesion and platelet-derived growth factor. Such phosphorylation of SPIN90 likely promotes the interaction of the $SPIN90{\cdot}{\beta}PIX{\cdot}WASP$ complex and Nck. It thus appears that the interaction of the $SPIN90{\cdot}{\beta}PIX{\cdot}WASP$ complex with Nck is crucial for stable cell adhesion and can be dynamically modulated by SPIN90 phosphorylation that is dependent on cell adhesion and ERX activation. SPIN90 directly binds syndapin I, syndapin isoform II-1 and II-s via its PRD region in vitro, in vivo and also associates with endocytosis core components such as clathrin and dynamin. In neuron and fibroblast, SPIN90 colocalizes with syndapins as puntate form, consistent with a role for SPIN90 in clathrin-mediated endocytosis pathway. Overexpression of SPIN90 N-term inhibits receptor-mediated endocytosis. Interestingly, SPIN90 PRD, binding interface of syndapin, significantly blocks internalization of transferrin, demonstrating SPIN90 involvement in endocytosis in vivo by interacting syndapin. Depletion of endogenous SPIN90 by introducing $\alpha-SPIN90$ also blocks receptor-mediated endocytosis. Actin polymerization could generate farce facilitating the pinch-out event in endocytosis, detach newly formed endocytic vesicle from the plasma membrane or push out them via the cytosol on actin tails. Here we found that SPIN90 localizes to high actin turn over cortical area, actin-membrane interface and membrane ruffle in PDGF treated cells. Overexpression of SPIN90 has an effect on cortical actin rearrangement as filopodia induction and it is mediated by the Arp2/3 complex at cell periphery. Consistent with a role in actin organization, CFP-SPIN90 present in actin comet tail generated by PIP5 $kinase\gamma$ overexpression. Therefore this study suggests that SPIN90 is functional linker between endocytosis and actin cytoskeleton.

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