• The Journal of the Institute of Internet, Broadcasting and Communication
• /
• v.13 no.2
• /
• pp.149-155
• /
• 2013

In these days, Entity-Relationship Model is the most popular modeling tool for designing databases, and XML is a de facto standard language for representing and exchanging data. But, because of many commercial products supporting Entity-Relationship Model use their's own representation formats, and thus it gives rise to difficulties the inter-operability between these products. In this paper, we propose an ERX, a generation tool of XML Schema from Entity-Relationship Model. ERX receives an Entity-Relationship Diagram as an input, transforms it based on transformation rules, and generates a XML Schema Definition as an output. Transformation rules contain entity set, relationship set, mapping cardinalities, and generalization.

• Digital Contents
• /
• no.1 s.56
• /
• pp.48-53
• /
• 1998

• ETRI Journal
• /
• v.43 no.3
• /
• pp.389-399
• /
• 2021

In this paper, we propose one-bit feedback-based distributed beamforming (DBF) techniques for simultaneous wireless information and power transfer in interference channels where the information transfer and power transfer networks coexist in the same frequency spectrum band. In a power transfer network, multiple distributed energy transmission nodes transmit their energy signals to a single energy receiving node capable of harvesting wireless radio frequency energy. Here, by considering the Internet-of-Things sensor network, the energy harvesting/information decoding receivers (ERx/IRx) can report their status (which may include the received signal strength, interference, and channel state information) through one-bit feedback channels. To maximize the amount of energy transferred to the ERx and simultaneously minimize the interference to the IRx, we developed a DBF technique based on one-bit feedback from the ERx/IRx without sharing the information among distributed transmit nodes. Finally, the proposed DBF algorithm in the interference channel is verified through the simulations and also implemented in real time by using GNU radio and universal software radio peripheral.

• Proceedings of the Microbiological Society of Korea Conference
• /
• 2004.05a
• /
• pp.141-146
• /
• 2004

SPIN90 was identified to farm molecular complex with $\betaPIX$, WASP and Nck. This complex shows that SPIN90 interacts with Nck in a manner dependent upon cell adhesion to extracellular matrix, but $SPIN90{\cdot}{\beta}PIX{\cdot}WASP$ complex was stable even in suspended cells. This suggests that SPIN90 serves as an adaptor molecule to recruit other proteins to Nck at focal adhesions. SPIN90 was phosphorylated by ERK1, which was, itself, activated by cell adhesion and platelet-derived growth factor. Such phosphorylation of SPIN90 likely promotes the interaction of the $SPIN90{\cdot}{\beta}PIX{\cdot}WASP$ complex and Nck. It thus appears that the interaction of the $SPIN90{\cdot}{\beta}PIX{\cdot}WASP$ complex with Nck is crucial for stable cell adhesion and can be dynamically modulated by SPIN90 phosphorylation that is dependent on cell adhesion and ERX activation. SPIN90 directly binds syndapin I, syndapin isoform II-1 and II-s via its PRD region in vitro, in vivo and also associates with endocytosis core components such as clathrin and dynamin. In neuron and fibroblast, SPIN90 colocalizes with syndapins as puntate form, consistent with a role for SPIN90 in clathrin-mediated endocytosis pathway. Overexpression of SPIN90 N-term inhibits receptor-mediated endocytosis. Interestingly, SPIN90 PRD, binding interface of syndapin, significantly blocks internalization of transferrin, demonstrating SPIN90 involvement in endocytosis in vivo by interacting syndapin. Depletion of endogenous SPIN90 by introducing $\alpha-SPIN90$ also blocks receptor-mediated endocytosis. Actin polymerization could generate farce facilitating the pinch-out event in endocytosis, detach newly formed endocytic vesicle from the plasma membrane or push out them via the cytosol on actin tails. Here we found that SPIN90 localizes to high actin turn over cortical area, actin-membrane interface and membrane ruffle in PDGF treated cells. Overexpression of SPIN90 has an effect on cortical actin rearrangement as filopodia induction and it is mediated by the Arp2/3 complex at cell periphery. Consistent with a role in actin organization, CFP-SPIN90 present in actin comet tail generated by PIP5 $kinase\gamma$ overexpression. Therefore this study suggests that SPIN90 is functional linker between endocytosis and actin cytoskeleton.