• 전자공학회논문지
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• 제50권1호
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• pp.21-26
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• 2013

본 논문에서는 그룹(group)을 2개의 심볼(symbol)로 나누는 시스템을 제안하는데, 이 두 개의 더해진 심볼들은 다중화에 의해 분리된 후 역다중화 기술을 이용해 다시 합해진다. 제안된 시스템에서 간단한 PIC(Partial Interference Cancelation) 그룹 복호화 기술은 Double-ABBA(D-ABBA) 유사 직교(Quasi-Orthogonal) 시공간 부호(Space Time Code)를 위해 사용된다. 이 부호는 고차 MIMO(Multiple Input Multiple Output) 시공간 블록 부호화에서 복호화시에 복잡도를 줄여준다. 그리고 기존의 방법과 제안된 방법과의 성능을 비교한다.

• Journal of Preventive Medicine and Public Health
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• 제47권1호
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• pp.57-63
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• 2014

Objectives: We established a Wistar rat model of asthma caused by toluene diisocyanate (TDI) exposure, and investigated the relationship between TDI exposure concentrations and respiratory hypersensitivity, airway inflammation, and cytokine secretions in animals, to better understand the mechanism of TDI induced occupational asthma. Methods: Wistar rats were exposed to two different concentrations of TDI vapor four hours a day for five consecutive days. Bronchoalveolar lavage (BAL) was performed, and differential leucocytes from the BAL fluid were analyzed. Lung histopathological examination was carried out to investigate the inflammatory status in the airways. Production of cytokines interleukin (IL)-4 and IL-5 productions in the BAL fluid in vivo was determined with enzyme-linked immunosorbent assay kits. Results: The TDI-exposed rats exhibited greater airway hypersensitivity symptoms than the control rats. The BAL differential cell count and lung histopathological examination demonstrated that inflammation reactions were present in both the central and peripheral airways, characterized with marked infiltration of eosinophils in the TDI-exposed rats. The cytokine assay showed that IL-4 and IL-5 were predominantly produced in the BAL fluid in vivo. Conclusions: These findings imply that TDI exposure concentrations may greatly affect the occurrence and extent of inflammatory events and that Th2 type cytokines may play an important role in the immunopathogenesis of TDI-induced occupational respiratory hypersensitivity.