• Proceedings of the KIEE Conference
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• 1999.07f
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• pp.2712-2714
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• 1999

The small signal model for input series-output parallel connected converter system employing charge control together with input capacitor voltage feedback loop is developed. From the model developed, the effect of input capacitor voltage feedback loop to the system stability and outer loop compensator design is analyzed. Theoretical results and simulation show that input capacitor voltage feedback loop has no critical effects on the system stability, so the system can be reduced to a equivalent single module for the stability analysis and outer loop compensator design.

• Proceedings of the KIEE Conference
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• 2003.07a
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• pp.212-214
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• 2003

A passive loop is considered to be a cheap and easy way to be realized among the magnetic field mitigation methods. So this paper evaluates and quantizes systematically the effectiveness of a passive loop on mitigating magnetic field(M-field) and examines the feasibility and problems in adoption. To do so, first, we explain the principles of M-field mitigation through a passive loop and derive formulas for 2-D M-field analysis. Next this paper simulates the M-field mitigation patterns for a flat type 1-circuit transmission line installed a passive loop.

• Proceedings of the Korean Magnestics Society Conference
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• 1997.11a
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• pp.128-129
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• 1997

• Proceedings of the PSK Conference
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• 2000.04a
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• pp.140.2-140.2
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• 2000

• Proceedings of the Korean Nuclear Society Conference
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• 1999.10a
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• pp.164-164
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• 1999

• Journal of the Korean Ceramic Society
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• v.19 no.4
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• pp.267-274
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• 1982

The sintering behavior and B-H loop properties of $Li_{0.5O4}$ with the addition of $Nb_2O_5$ and $V_2O_5$ have been investigated by observation of microstructures and measurement of semi-static hysteresis loops. The sintering temperature was lowered by the additions of $Nb_2O_5$ and $V_2O_5$ and the effect of $V_2O_5$ addition was greater than that of $Nb_2O_5$. The abnormal grain growth was observed at about 100$0^{\circ}C$(d:4.6g/㎤) and 85$0^{\circ}C$(d:4.5g/㎤) in the specimens with the addition of $Nb_2O_5$ and $V_2O_5$ respectively. The addition of $Nb_2O_5$ retulted in a large and uniform grain size, and the addition of $V_2O_5$ resulted in a uniform grain size but the final density was lower than that of the pure specimen. The squareness was increased and the coercive force was decreased by the addition of $Nb_2O_5$, and the squareness was decreased and the coercive force was increased by the addition of $V_2O_5$. These effects could be explained by a large and uniform grain size, and by a lower final density of specimens with the addition of $Nb_2O_5$ and $V_2O_5$ respectively.

• Proceedings of the Korean Biophysical Society Conference
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• 2003.06a
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• pp.69-69
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• 2003

Human CD99 is a ubiquitous 32-kDa transmembrane protein encoded by the mic2 gene. The major cellular functions of CD99 protein are related to homotypic cell adhension, apoptosis, vesicular protein transport, and differentiation of thymocytes or T cells. Recently it has been reported that expression of a splice variant of CD99 transmembrane protein (Type I and Type II) increases invasive ability of human breast cancer cells. To understand structural basis for cellular functions of CD99 (Type I), we have initiated studies on hCD99$^{TMcytoI}$ and hCD99$^{cytoI}$ using circular dichroism (CD) and multi-dimensional NMR spectroscopy. CD spectrum of hCD99$^{TMcytoI}$ in the presence of 200mM DPC and CHAPS displayed an existence $\alpha$-helical conformation. The solution structure of hCD99$^{cytoI}$ determined by NMR is composed of one N-terminal $\alpha$-helix, $\alpha$A, two C-terminal short $\alpha$-helix segments, $\alpha$B and $\alpha$C. While $\alpha$A and $\alpha$B are connected by the long flexible loop, $\alpha$B and $\alpha$C connected by type III$\beta$-turn. Although it has been rarely figured out the correlation between structure and functional mechanism of hCD99$^{TMcytoI}$ and hCD99$^{cytoI}$, there is possibility of dimerization or oligomerization. In addition, the feasible mechanism of hCD99$^{cytoI}$ is that it could have intramolecular interaction between the N- and C- terminal domain through large flexible AB loop.

• Proceedings of the KIPE Conference
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• 2004.07b
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• pp.583-586
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• 2004

A typical method to control the single-phase power converter system is to utilize the zero-crossing PLL. However, this method is vulnerable to the voltage disturbance and affects the performance of controller This paper proposes a new single-phase PLL system that is composed of the adaptive linear combiner and the PI control. The operational principle was analyzed through theoretical approach and the performance was verified through simulations with MATLAB. The proposed PLL system shows rapidness and robustness in control under the voltage disturbances such as the sag, harmonics, and phase jump.

• Structural Engineering and Mechanics
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• v.15 no.2
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• pp.239-248
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• 2003

The dynamic response analysis of closed loop control system based on probability for the intelligent truss structures with random parameters is presented. The expressions of numerical characteristics of structural dynamic response of closed loop control system are derived by means of the mode superposition method, in which the randomness of physical parameters of structural materials, geometric dimensions of active bars and passive bars, applied loads and control forces are considered simultaneously. The influences of the randomness of them on structural dynamic response are inspected by several engineering examples and some significant conclusions are obtained.

• Journal of Ginseng Research
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• v.45 no.6
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• pp.734-743
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• 2021

Background: The underlying mechanisms of the potential tumor-suppressive effects of ginsenoside Rh2 are complex. N6-methyladenosine (m6A) RNA methylation is usually dysregulated in cancer. This study explored the regulatory effect of ginsenoside Rh2 on m6A RNA methylation in cancer. Methods: m6A RNA quantification and gene-specific m6A RIP-qPCR assays were applied to assess total and gene-specific m6A RNA levels. Co-immunoprecipitation, fractionation western blotting, and immunofluorescence staining were performed to detect protein interactions and distribution. QRT-PCR, dual-luciferase, and ChIP-qPCR assays were conducted to check the transcriptional regulation. Results: Ginsenoside Rh2 reduces m6A RNA methylation and KIF26B expression in a dose-dependent manner in some cancers. KIF26B interacts with ZC3H13 and CBLL1 in the cytoplasm of cancer cells and enhances their nuclear distribution. KIF26B inhibition reduces m6A RNA methylation level in cancer cells. SRF bound to the KIF26B promoter and activated its transcription. SRF mRNA m6A abundance significantly decreased upon KIF26B silencing. SRF knockdown suppressed cancer cell proliferation and growth both in vitro and in vivo, the effect of which was partly rescued by KIF26B overexpression. Conclusion: ginsenoside Rh2 reduces m6A RNA methylation via downregulating KIF26B expression in some cancer cells. KIF26B elevates m6A RNA methylation via enhancing ZC3H13/CBLL1 nuclear localization. KIF26B-SRF forms a positive feedback loop facilitating tumor growth.