• The Journal of Society for e-Business Studies
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• v.14 no.4
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• pp.315-332
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• 2009

In logistic environments, a process, in that it manages the flow of materials among partners, involves more than one organization. In this regard, a logistic process, as a combined process consisting of multiple sub processes, needs to be managed with controling interaction among partners. In achieving systematic management of a logistic process, traditional Business Process Management (BPM) cannot be used for the entire flow, since it lacks the ability to manage interactions among partners. Particularly in logistic environments where RFID technologies are used, how to deal with the connection between RFID event and logistic flow has not been properly addressed. To overcome this limitation, this paper proposes a new method of managing multi-organizational logistic processes based on RFID events. We define inter-workflow pattern, and suggest ECA(Event-Condition-Action) rules for auto triggering of logistic processes. To adjust the rules to RFID events, we invent RFID-based ECA rules using complex event. A prototype system has been developed for the purpose of demonstrating the effectiveness of our approach.

• Journal of Auto-vehicle Safety Association
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• v.15 no.4
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• pp.71-78
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• 2023

This study presents the direction of data triggers and elements to be recorded in automated vehicles in the future in relation to the event data recorder (EDR) and data storage system for automated driving (DSSAD). It does not distinguish between the EDR and DSSAD, but suggests data triggers and elements in preparation for overall automated vehicle accidents and dangerous situations. To propose, the current status of discussions on EDR/DSSAD internationally and the case of investigating accidents with automated vehicles under temporary driving licenses in Korea were analyzed. Based on the analysis, the direction of data triggers and elements of the EDR/DSSAD of automated vehicles were presented.

• The Transactions of The Korean Institute of Electrical Engineers
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• v.61 no.8
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• pp.1179-1185
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• 2012

This paper presents a new approach for the detection of seismic events using accumulated changes on time-frequency domain and variable threshold. To detect seismic P-wave arrivals with rapidness and accuracy, it is that the changes on the time and the frequency domains are simultaneously used. Their changes are parameters appropriated to reflect characteristics of earthquakes over moderate magnitude(${\geq}$ magnitude 4.0) and microearthquakes. In addition, adaptively controlled threshold values can prevent false P-wave detections due to low SNR. We tested our method on real earthquakes those have various magnitudes. The proposed algorithm gives a good detection performance and it is also comparable to STA/LTA algorithm in computational complexity. Computer simulation results shows that the proposed algorithm is superior to the conventional popular algorithm (STA/LTA) in the seismic P-wave detection.

• IMMUNE NETWORK
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• v.14 no.1
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• pp.7-13
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• 2014

Molecular mimicry is an attractive mechanism for triggering autoimmunity. In this review, we explore the potential role of evolutionary conserved bacterial proteins in the production of autoantibodies with focus on granulomatosis with polyangiitis (GPA) and rheumatoid arthritis (RA). Seven autoantigens characterized in GPA and RA were BLASTed against a bacterial protein database. Of the seven autoantigens, proteinase 3, type II collagen, binding immunoglobulin protein, glucose-6-phosphate isomerase, ${\alpha}$-enolase, and heterogeneous nuclear ribonuclear protein have well-conserved bacterial orthologs. Importantly, those bacterial orthologs are also found in human-associated bacteria. The wide distribution of the highly conserved stress proteins or enzymes among the members of the normal flora and common infectious microorganisms raises a new question on how cross-reactive autoantibodies are not produced during the immune response to these bacteria in most healthy people. Understanding the mechanisms that deselect auto-reactive B cell clones during the germinal center reaction to homologous foreign antigens may provide a novel strategy to treat autoimmune diseases.

• Journal of the Korean Society of Safety
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• v.28 no.3
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• pp.51-55
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• 2013

The purpose of this study is to find the causes of malfunctions and defective operation of printed circuit boards(PCBs) built into home refrigerators to perform power saving functions. This study performed an electrostatic test of a PCB built-in using an Auto Triggering system; lightning and impulse tests using an LSS-15AX; and an impulse test using an INS-400AX. From the analysis of a secondarily developed product, it was found that electrostatic discharge(ESD) caused more malfunctions and defective operations than electric overstress(EOS) due to overvoltage. As a result of increasing the condenser capacity of the PCB circuit, withstanding voltage was increased to 7.4 kV. In addition, this study changed the power saving mode and connected a varistor to the #2 pin of an IC chip. As a result, the system consisting of all specimens of a finally developed product was operated stably with an applied voltage of less than 10 kV. This study found it necessary to perform quality control at the manufacturing stage in order to reduce the occurrence of electrostatic accidents to IC chips built into a PCB.

• Journal of Physiology & Pathology in Korean Medicine
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• v.16 no.5
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• pp.914-920
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• 2002

This experiment is carried out for test whether the addition temperament drugs of Danchisoyo-san have an anti-inflammatory effect and have suppression effect on immunocyte in the state of inflammation which induced by carageenan and zymosan. The freezing lyophilization powder which were extracted from Danchisoyo-san divided low dose group(200mg/kg-DSL) and high dose group(500mg/kg-DSH) and after melting in water, it was orally administered to the mouse. Compared with inflammation induced group which were induced by triggering-inflammation reagent carageenan and zymosan and normal contrast group, we measured the edema decrement effect, macrophage and spleen cell activation. 1. Danchisoyo-san has suppress inflammatory reaction induced by carrageenan. 2. Danchisoyo-san has suppress increasing activation of abdominal cavity macrophage cell in the carrageenan induced inflammation. 3. Danchisoyo-san has suppress increasing activation of spleen cell in the carrageenan induced inflammation. 4. Danchisoyo-san has suppress increasing activation of abdominal cavity macrophage in the zymosan induced inflammation. 5. Danchisoyo-san has suppress increasing activation of spleen cell in the zymosan induced inflammation. Based on the above result, Danchisoyo-san was improved its suppression effect to the inflammatory reaction through the suppression of spleen cell and macrophage activation. So we concluded that Danchisoyo-san is prospected as a anti-inflammatory agent to cure inflammation.

• Archives of Pharmacal Research
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• v.22 no.5
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• pp.437-447
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• 1999

Type I diabetes, also known as insulin-dependent diabetes mellitus (IDDM) results from the destruction of insulin-producing pancreatic $\beta$ cells by a progressive $\beta$ cell-specific autoimmune process. The pathogenesis of autoimmune IDDM has been extensively studied for the past two decades using animal models such as the non-obese diabetic (NOD) mouse and the Bio-Breeding (BB) rat. However, the initial events that trigger the immune responses leading to the selective destruction of the $\beta$ cells are poorly understood. It is thought that $\beta$ cell auto-antigens are involved in the triggering of $\beta$ cell-specific autoimmunity. Among a dozen putative $\beta$ cell autoantigens, glutamic acid decarboxylase (GAD) has bee proposed as perhaps the strongest candidate in both humans and the NOD mouse. In the NOD mouse, GAD, as compared with other $\beta$ cell autoantigens, provokes the earliest T cell proliferative response. The suppression of GAD expression in the $\beta$ cells results in the prevention of autoimmune diabetes in NOD mice. In addition, the major populations of cells infiltrating the iselts during the early stage of insulitis in BB rats and NOD mice are macrophages and dendritic cells. The inactivation of macrophages in NOD mice results in the prevention of T cell mediated autoimmune diabetes. Macrophages are primary contributors to the creation of the immune environment conducive to the development and activation of $\beta$cell-specific Th1-type CD4+ T cells and CD8+ cytotoxic T cells that cause autoimmune diabetes in NOD mice. CD4+ and CD8+ T cells are both believed to be important for the destruction of $\beta$ cells. These cells, as final effectors, can kill the insulin-producing $\beta$ cells by the induction of apoptosis. In addition, CD8+ cytotoxic T cells release granzyme and cytolysin (perforin), which are also toxic to $\beta$ cells. In this way, macrophages, CD4+ T cells and CD8+ T cells act synergistically to kill the $\beta$ cells in conjunction with $\beta$ cell autoantigens and MHC class I and II antigens, resulting in the onset of autoimmune type I diabetes.