• Smart Media Journal
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• v.12 no.2
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• pp.56-65
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• 2023

In broadcasts such as news and coverage programs, voice is modulated to protect the identity of the informant. Adjusting the pitch is commonly used voice modulation method, which allows easy voice restoration to the original voice by adjusting the pitch. Therefore, since broadcast voice modulation methods cannot properly protect the identity of the speaker and are vulnerable to security, a new voice modulation method is needed to replace them. In this paper, using the Lightweight speech de-identification model as the evaluation target model, we compare speech de-identification performance with broadcast voice modulation method using pitch modulation. Among the six modulation methods in the Lightweight speech de-identification model, we experimented on the de-identification performance of Korean speech as a human test and EER(Equal Error Rate) test compared with broadcast voice modulation using three modulation methods: McAdams, Resampling, and Vocal Tract Length Normalization(VTLN). Experimental results show VTLN modulation methods performed higher de-identification performance in both human tests and EER tests. As a result, the modulation methods of the Lightweight model for Korean speech has sufficient de-identification performance and will be able to replace the security-weak broadcast voice modulation.

• Proceedings of the Korean Institute of Information and Commucation Sciences Conference
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• 2017.10a
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• pp.552-555
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• 2017

It is de-identification that emerged to find the trade-off between the use of big data and the protection of personal information. In particular, in the field of medical that deals with various semi-identifier information and sensitive information, de-identification must be performed in order to use medical consultation such as EMR and voice, KakaoTalk, and SNS. However, there is no separate law for medical information protection and legislation for de-identification. Therefore, in this study, we present the current status of de-identification of personal information, the status and case of de-identification of medical information, and finally we provide issues and solutions for medial information protection and de-identification.

• Proceedings of the Acoustical Society of Korea Conference
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• 1998.06e
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• pp.327-330
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• 1998

독립성분분석(ICA: Independent Component Analysis)이란 특징이 상이한 둘 이상의 신호들이 선형적으로 결합되어 있을 때 이를 효과적으로 분리하는 방법들을 통칭하며 잡음제거, 음질개선 및 신호처리 분야에서 많이 활용되고 있다. 본 논문에서는 전화음성 화자인식 시스템의 성능향상을 위해 독립성분분석을 이용하는 방법을 제안한다. 먼저 화자가 발성한 음성신호의 켑스트럼 계수를 여러 채널 함수들의 선형적인 합으로 가정하고, 독립성분분석을 이용하여 얻은 새로운 켑스트럼 벡터를 학습과 인식에 사용하였다. 실험자료는 잔화음성 화자식별기의 성능평가에 널리 쓰이고 있는 SPIDRE를 사용하였고 regodic 은닉 마코프 모델을 이용하여 문장 독립 화자식별 시스템을 구성하였다. 학습음성의 특징과 실험음성의 특징이 다른 조건에서 기존의 채널 정규화 방법들에 비해 10~15%이상 인식률이 향상되었다.

• 한국HCI학회:학술대회논문집
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• 2007.02a
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• pp.293-297
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• 2007

Human Computer Interaction 기술을 구체화 시키기 위한 Intelligent Responsive Space의 개발에 있어서 음성정보는 여러 가지로 유용하게 활용될 수 있다. 음성신호로부터 얻을 수 있는 다양한 정보 중의 하나가 화자인식을 이용한 화자의 신원식별이다. 이 논문에서는 화자인식 인식이 어려운 환경에서도 음성 신호로부터 추출한 특성벡터들을 선택적으로 사용함으로써 화자인식 성능을 높일 수 있는 새로운 방법을 제안하려 한다. 화자를 인식하는데 있어서 인식오류를 발생시킬 가능성이 높은 특성벡터들을 인식을 위한 판단의 대상에서 배제시킴으로써 성능을 향상시킬 수 있다. 실험결과에 의하면 0.25초에서2초 길이의 짧은 음성만으로도 기존의 방법에 비해 20에서 51%의 상대적 성능 향상을 보였다. 새롭게 제안된 방법을 적용하면 기존의 방법들에 비해 세밀하면서도 정확하게 연속적으로 화자들을 인식할 수 있게 된다.

• The KIPS Transactions:PartC
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• v.9C no.4
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• pp.459-466
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• 2002

This paper proposes a robust speaker identification algorithm based on the wavelet filter banks and multiple decision-making scheme. Since the proposed speaker identification algorithm has a structure performing the identification algorithm independently for each subband, the noise effect of an subband can be localized. Through this process, we can obtain more robust results for the environmental noises which generally have band limited frequency. In the experiments, the proposed method showed more 15∼60% improvement than the vector quantization method for the various noisy environments.

• The KIPS Transactions:PartB
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• v.8B no.2
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• pp.189-194
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• 2001

본 논문에서는 개인 식별을 위하여 복수 해상도 구조를 제시하였고 이 방법으로 구순문 인식을 구현하였다. 구순문 인식은 지문, 음성 패턴, 홍채 패턴과 얼굴 인식과 같은 신체적 특징에 비하여 상대적으로 연구가 많이 이루어지지 않은 신체적 특징이다. 구순문은 CCD 카메라를 이용할 경우 홍채나 얼굴 패턴 같은 다른 특징 요소와 연결하여 인식 시스템을 구축할 수 있는 장점을 가지고 있다. 구순문 인식을 위해 pattern kernels를 이용한 새로운 방법을 제시하였다. Pattern kernels는 여러 개의 local lip print mask들로 구성된 함수이며, lip print의 정보를 디지털 데이터로 전환시켜 준다. 복수 해상도를 가지는 인식 시스템은 단일 해상도의 시스템보다 더욱 신뢰적이며 인식률도 높다.

• Journal of Life Science
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• v.32 no.11
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• pp.833-840
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• 2022

Chronic inflammation has been shown to be closely associated with tumor development and progression. Nuclear factor kappa B (NF-κB) is composed of a family of five transcription factors. NF-κB signaling plays a crucial role in the inflammatory response and is often found to be dysregulated in various types of cancer, making it an attractive target in cancer therapeutics. In this study, CDC-like kinase 3 (CLK3) was identified as a novel kinase that regulates the NF-κB signaling pathway. Our data demonstrate that CLK3 inhibits the canonical and non-canonical NF-κB pathways. Luciferase assays following the transient or stable expression of CLK3 indicated that this kinase inhibited NF-κB activation mediated by Tumor necrosis factor-alpha (TNFα) and Phorbol 12-myristate 13-acetate (PMA), which are known to activate NF-κB signaling via the canonical pathway. Consistent with data on the ectopic expression of CLK3, CLK3 knockdown using shRNA constructs increased NF-κB activity 1.5-fold upon stimulation with TNFα in HEK293 cells compared with the control cells. Additionally, overexpression of CLK3 suppressed the activation of this signaling pathway induced by NF-κB-inducing kinase (NIK) or CD40, which are well-established activators of the non-canonical pathway. To further examine the negative impact of CLK3 on NF-κB signaling, we performed Western blotting following the TNFα treatment to directly identify the molecular components of the NF-κB pathway that are affected by this kinase. Our results revealed that CLK3 mitigated the phosphorylation/activation of transforming growth factor-α-activated kinase 1 (TAK1), inhibitor of NF-κB kinase alpha/beta (IKKα/α), NF-κB p65 (RelA), NF-κB inhibitor alpha (IκBα), and Extracellular signal-regulated kinase 1/2-Mitogen-activated protein kinase (ERK1/2-MAPK), suggesting that CLK3 inhibits both the NF-κB and MAPK signaling activated by TNFα exposure. Further studies are required to elucidate the mechanism by which CLK3 inhibits the canonical and non-canonical NF-κB pathways. Collectively, these findings reveal CLK3 as a novel negative regulator of NF-κB signaling.