• Title/Summary/Keyword: 신장병

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지혜 깊어지는 건강: 40대 몸을 지키는 요가 -당뇨병, 요가로 예방하자

  • Lee, Gyeong-Hui
    • 건강소식
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    • v.35 no.4
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    • pp.23-23
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    • 2011
  • 운동을 하게 되면 높아진 혈당이 떨어지게 된다. 이는 운동이 열량을 소모시키고 근육으로 혈액 순환을 증가시키며 혈당을 낮추는 호르몬인 인슐린의 작용을 도와주기 때문이다. 단, 당뇨병으로 인한 신장의 합병증이 있을 때에는 심한 운동이 오히려 해가 될 수 있으니 운동 강도를 조절해야 한다.

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당뇨합병증 : 생소한 당뇨합병증 - 당뇨인의 이비인후과 질환

  • Kim, Seon-Ju
    • The Monthly Diabetes
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    • s.259
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    • pp.40-41
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    • 2011
  • 당뇨는 우리 몸 구석구석에 영향을 주어 다양한 증상을 불러일으킨다. 당뇨병이 눈, 혈관, 신장 등에 영향을 준다는 것은 다들 아는 사실이나 이비인후과적 합병증은 다소 생소할 수 있다. 하지만 당뇨병이 난청, 악성 외이도염, 수면무호흡증 등을 발병시킬 수도 있다. 당뇨관리의 부주의가 어떤 이비인후과적 질환을 가져오는지 알아보자.

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A Case of Progressive FSGS and Chronic Kidney Disease in Congenital Chloride Diarrhea with SLC26A3 Mutation (선천성 염소성 설사를 가진 환아에서 국소 분절 사구체경화증이 발생하여 만성 신장병으로 발전한 사례)

  • Seo, Young-Jun;Cheong, Han Bin;An, Seok Min;Sin, Woo Cheol;Bae, Eun Joo;Yoon, Jong Hyung;Jeong, Hwal Rim;Lee, Hong Jin
    • Journal of The Korean Society of Inherited Metabolic disease
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    • v.18 no.3
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    • pp.87-94
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    • 2018
  • We present the case of long-term observation of a patient with chronic kidney disease (CKD) caused by advanced focal segmental glomerulosclerosis (FSGS) resulting from underlying congenital chloride diarrhea (CLD). A 20-year-old woman was admitted for prolonged proteinuria despite conservative treatment for CLD. She was diagnosed with CLD and started taking KCl salt supplementation from the time of birth. Mild proteinuria was first found at 12 years of age, which progressed to moderate proteinuria at 16 years of age. At 16 years of age, CKD stage 2 with FSGS was diagnosed based on the initial assessment of the glomerular filtration rate (GFR) and kidney histology. On admission, we re-assessed her renal function, histology and genetic analysis. GFR had deteriorated to CKD stage 4 and renal histology revealed an advanced FSGS combined with tubulointerstitial fibrosis. A homozygous mutation in the SLC26A3 gene (c.2063-1G>T) was found by diagnostic exome sequencing and may have been inherited from both parents. CLD patients can be more vulnerable to renal injury, which may also cause progression of renal failure. Therefore, even if there is an early diagnosis and adequate salt supplementation, close monitoring of renal function and tailored treatment should be emphasized for renal protection and favorable CLD prognosis.

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A Case of Microscopic Polyangiitis Initially Suspected with Henoch-Sch$\ddot{o}$nlein Purpura Nephritis (Henoch-Sch$\ddot{o}$nlein 자반병 신염으로 의심했던 현미경적 다발혈관염 1례)

  • Im, Jong Geun;Moon, Kyung Chul;Koo, Ja Wook
    • Childhood Kidney Diseases
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    • v.16 no.2
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    • pp.132-137
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    • 2012
  • Microscopic polyangiitis (MPA) is systemic small vessel vasculitis that is very rare in childhood. MPA is characterized by pauci-immune necrotizing small vessel vasculitis without clinical or pathological evidence of necrotizing granulomatous inflammation. Approximately 90% of patients have glomerulonephritis that is accompanied by a variety of other organ involvement. A 10-year-old girl visited our clinic with clinical manifestations suggestive Henoch-Sch$\ddot{o}$nlein purpura nephritis such as purpuric skin rash, abdominal pain, arthralgia on both knees, massive proteinuria and microscopic hematuria. So initially we suspected Henoch-Sch$\ddot{o}$nlein purpura nephritis. However, later her perinuclear-antineutrophil cytoplasmic antibodies(p-ANCA) test was positive, and her renal biopsy was consistent with microscopic polyangiitis. We began steroid therapy, combined with cyclophosphamide, ACE inhibitor. Currently she is a 12-year old, and until now she has been regularly examined in the outpatient. We report a case of microscopic polyangiitis initially suspected with Henoch-Sch$\ddot{o}$nlein purpura nephritis.

Application of Signal Processing for Kidney Disease Diagnosis (신장 질환 진단을 위한 신호처리기법의 적용)

  • Cho, Dong-Uk;Lee, Se-Hwan;Kim, Bong-Hyun
    • Proceedings of the Korea Information Processing Society Conference
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    • 2005.11a
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    • pp.651-654
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    • 2005
  • 신장 질환은 고혈압과 당뇨등에 대한 합병증으로 많이 발생되며 병이 상당 부분 진행이 되어야만 자각 증세가 있는 질환이다. 따라서 신장 질환은 예방이 중요하며 이를 위한 기술적 방법론이 개발되어야 한다. 현재는 소변 검사를 통한 단백뇨와 혈액 검사에 의지하여 질환의 유무와 진행 정도를 파악하고 있지만 이를 생체 신호 분석을 통해 행한다면 질환 예방등에 유익한 방법이 되리라 여겨진다. 본 논문에서는 이를 위해 신호 처리 기법을 적용하여 신장 질환을 판단하는 방법론을 제안하고자 한다. 이를 위해 음성 신호를 분석하고 신장과 관련이 되어 있는 오관인 귀에 대한 영역을 추출하는 방법을 제안하고자 한다. 끝으로 실험에 의해 제안한 방법의 유용성을 입증하고자 한다.

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Evaluation of Cerebral Blood Flow Using Arterial Spin Labeling in Patients with Chronic Kidney Disease (만성 콩팥병 환자들에서 동맥 스핀 표지 기법을 이용한 뇌 관류상태의 평가)

  • Se Won Oh;Samel Park;Nam-jun Cho;Hyo-Wook Gil;Eun Young Lee;Hyung Geun Oh;Sung-Tae Park
    • Journal of the Korean Society of Radiology
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    • v.81 no.4
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    • pp.912-919
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    • 2020
  • Purpose This study aimed to compare the brain perfusion status of patients with chronic kidney disease to a normal control group to identify any significant differences. Materials and Methods The perfusion state of the brain was measured by MRI using the arterial spin labeling technique in 36 patients undergoing hemodialysis due to chronic kidney disease and 36 normal controls. Images were then analyzed in a voxel-wise manner to detect brain areas showing significant perfusion differences between the two groups. Results Patients with chronic kidney disease showed increased perfusion in the form of large clusters across the right fronto-parieto-temporal lobe and the left parieto-occipital lobe. In addition, perfusion increased in the bilateral thalami, midbrain, pons, and cerebellum (p < 0.01, familywise error corrected). Conclusion Brain perfusion appears to increase in patients with chronic kidney disease compared to normal controls. Uremic toxicity is thought to be the cause of this increase as it can cause damage to the microscopic blood vessels and their surrounding structures.

HYPOPHOSPHATEMIC RICKETS : CASE REPORT (저인산혈증성 구루병 환아의 증례 보고)

  • Park, Yoon-Hee;Choi, Byung-Jai;Lee, Jong-Gap
    • Journal of the korean academy of Pediatric Dentistry
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    • v.27 no.1
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    • pp.108-112
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    • 2000
  • Hypophosphatemic rickets is lack of reponse to physiologic doses of vitamin D, different from the vitamin D-dependent rickets. It is inherited in an X-linked dominant fashion. The prime features of this disorder are lowered serum phosphate levels, elevated serum alkaline phosphatase and normal serum calcium levels. The dental manifestation often include apical radiolucencies, abscess and fistulas of clinically sound teeth. Dental radiographs show ricketic bone trabeculations, abscent or abnormal lamina dura and abnormal cementum. This case which was diagnosed to hypophosphatemic rickets, showed multiple spontaneous periapical abscess and gingival fistula enlarged pulp chambers, extension of the pulp horns into the cusp tips and delayed eruption.

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