• Title/Summary/Keyword: 명성손상

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A Study on the Judgment Criteria for the Trademark Dilution of Famous Marks

  • Park, Jong-Ryeol;Noe, Sang-Ouk
    • Journal of the Korea Society of Computer and Information
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    • v.24 no.10
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    • pp.225-232
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    • 2019
  • The trademark dilution of famous marks as a kind of unfair competition practice is defined and regulated in Article 2 (1) (c) of the Unfair Competition Prevention and Trade Secret Protection Act (hereinafter referred to as the Unfair Competition Prevention Act), which was newly established according to the amendment of the Act on February 3, 2001. Famous trademarks are universally protected in all around the world, which are likewise protected in the Republic of Korea by the Unfair Competition Prevention Actin line with such international trends. In order to establish the trademark dilution of famous marks, it is necessary to have the following characteristics: (1) high reputation of the original mark, (2) use of identical or similar markscompared to the original mark, (3) occurrence of blurring of discrimination or tarnishment of reputation; in particular, with respect to the degree of proof of 'blurring of discrimination or tarnishment of reputation', which is a constituent requirement of the trademark dilution of famous marks, it is reasonable to interpret the trademark dilution as concrete endangerment offense, neither harm-based offense nor abstract endangerment offense, and thus it should be considered that the crime is established if a specific realistic risk of blurring of discrimination or tarnishment of reputation occurs. Furthermore, in relation to the specific criteria of 'blurring of discrimination or tarnishment of reputation', it is necessary to comprehensively judge the degree of individual behavior in specific matters as a normative factor as well as the psychosocial viewpoint of the general public.

Costly Litigation and the Pricing of Initial Public Offerings (소송위험이 최초공모주의 가격결정에 미치는 영향에 관한 연구)

  • Jang, Beom-Sik;Tinic Seha M.
    • The Korean Journal of Financial Management
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    • v.11 no.2
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    • pp.223-257
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    • 1994
  • 본 연구는 최초공모주식의 저가발행 원인에 대한 보험가설을 미국에서의 실제 소송사례를 중심으로 실증분석한다. Tinic(1988)은 최초공모주의 저가발행이 공모에 따르는 법률적 소송위험과 소송으로 인한 간사회사의 명예손상에 대한 일종의 보험효과를 가지고 있으며, 따라서 저가발행의 크기는 소송위험과 음의 관계에 있다고 주장한다. 또한 보험가설에 의하면 실제 소송이 발생했을 경우에도 저가발행의 크기는 소송결과에 유리하게 작용한다고 한다. 본 연구에서 는 1933-1990 기간동안 미국증권법 제 11조 조항을 위반하여 소송이 제기되었던 사례가운데 실제소송의 결과가 알려진 판례를 중심으로 보험가설의 주장을 실증분석 하였다. 또한 최초공모이후의 공모시장을 통한 기업자금 조달시에 주간사회사의 변경여부를 분석함으로써 소송으로 인한 간사회사의 명예손상의 정도를 파악하고자 했다. 연구결과에 따르면 투자자들에 의한 집단소송의 결과와 최초공모주의 저가발행의 크기에는 유의적인 관계가 있음이 실증적으로 제시되었다. 또한 간사회사의 명성과 실제 현금으로 환원한 소송위험간에도 유의적인 관계가 있음을 보여주고 있다. 한편 소송에 관련된 최초공모주의 경우에는 일단 소송이 제기된 후의 공모를 통한 추가기업자금 조달시에 기존의 간사회사를 전원 교체하는 등 통제집단과는 다른 형태를 보여 줌으로써 보험 가설의 주장을 지지하고 있다.

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Utility Analysis of Federated Learning Techniques through Comparison of Financial Data Performance (금융데이터의 성능 비교를 통한 연합학습 기법의 효용성 분석)

  • Jang, Jinhyeok;An, Yoonsoo;Choi, Daeseon
    • Journal of the Korea Institute of Information Security & Cryptology
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    • v.32 no.2
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    • pp.405-416
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    • 2022
  • Current AI technology is improving the quality of life by using machine learning based on data. When using machine learning, transmitting distributed data and collecting it in one place goes through a de-identification process because there is a risk of privacy infringement. De-identification data causes information damage and omission, which degrades the performance of the machine learning process and complicates the preprocessing process. Accordingly, Google announced joint learning in 2016, a method of de-identifying data and learning without the process of collecting data into one server. This paper analyzed the effectiveness by comparing the difference between the learning performance of data that went through the de-identification process of K anonymity and differential privacy reproduction data using actual financial data. As a result of the experiment, the accuracy of original data learning was 79% for k=2, 76% for k=5, 52% for k=7, 50% for 𝜖=1, and 82% for 𝜖=0.1, and 86% for Federated learning.

Effective of Body Temperature Increasing during Brain MRI scan (MRI 검사 시 체온상승 효과: 1.5 T vs 3.0 T)

  • Kim, Myeong Seong;Lee, Jongwoong;Jung, Jaeeun
    • Journal of the Korean Society of Radiology
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    • v.11 no.1
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    • pp.49-54
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    • 2017
  • As the Radiofrequency(RF) increases with the magnetic field strength, the wavelength of the RF excitation field becomes smaller, which leads to more the thermal effect in the human-body placed in the electric field. MRI scanner used was GE signa 1.5T, HDx 3.0T and Philips 3.0T with same routine clinical sequence protocol. Therefore temperature was measured before and after each scan. Taken the temperatures in the ear with ear infra-red type thermometer(Braun co). 3.0T were temperature increases more than $0.15^{\circ}C$ and GE 3.0T MRI equipment about $0.14^{\circ}C$ higher than the Philips 3.0T MRI(p<0.012). Psychogenic status was investigated by the survey respondents about their status can not just answer therefore, a little different from the expected. In our study of Thermal effect of clinical MRI with clinical protocol sequence, we found that the 3.0T in the body-temperature rise was greater than the 1.5T. Therefore, in clinical 3.0T examine the dangerous situation caused by the temperature rise occurred (burns, impaired thermoregulatory mechanism in patients with high-temperature damage, exhaustion occurs due to excessive sweating), not to appear the more watched the patient's condition with procedure.

Anti-Oxidative and Neuroprotective Effects of Rhei Rhizoma on BV-2 Microglia Cells and Hippocampal Neurons (대황(大黃)의 항산화와 신경세포손상 보호효능에 대한 연구)

  • Myung, Sung-Ha;Kim, Youn-Sub
    • Journal of Physiology & Pathology in Korean Medicine
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    • v.19 no.3
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    • pp.647-655
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    • 2005
  • This study demonstrated anti-oxidative and neuroprotective effects of Rhei Rhizoma. Anti-oxidative effects were studied on BV-2 microglia cells damaged by $H_2O_2$ and nitric oxide. Neuroprotective effects were studied by using oxygen/glucose deprivation of the organotypic hippocampal slice cultures. The results obtained are as follows; The groups treated with 0.5 and 5 mg/ml of Puerariae Radix revealed significant decreases of neuronal cell death area and cell death area percentages in CA1 region of ischemic damaged hippocampus cultures during whole 48 hours of the experiment. The group treated with 50 mg/ml of Puerariae Radix demonstrated decreases of neuronal cell death area and cell death area percentages in CA1 region, but these were not significant statistically. The groups treated with 0.5 and 5 mg/ml of Puerariae Radix revealed significant decreases of neuronal cell death area and cell death area percentages in dentate gyrus of ischemic damaged hippocampus cultures during whole 48 hours of the experiment. The group treated with 50 mg/ml of Puerariae Radix demonstrated decreases of neuronal cell death area and cell death area percentages in dentate gyrus, but these were not significant statistically. The groups treated with 0.5 and 5 mg/ml of Puerariae Radix revealed significant decreases of TUNEL-positive cells in both CA1 region and dentate gyrus of ischemic damaged hippocampus cultures. The group treated with 50 mg/ml of Puerariae Radix demonstrated significant decrease of TUNEL-positive cells in CA1 region, but not in dentate gyrus of ischemic damaged hippocampus. The groups treated with 0.5 and 5 mg/ml of Puerariae Radix revealed significant decreases of LDH concentrations in culture media of ischemic damaged hippocampus cultures. The group treated with 50 mg/ml of Puerariae Radix demonstrated decrease of LDH concentrations in culture media, but it was not significant statistically. The groups treated with 0.5 and 5 mg/ml of Puerariae Radix revealed significant increases of cell viabilities of BV-2 microglia cells damaged by $H_2O_2$. The group treated with 50 mg/ml of Puerariae Radix demonstrated increase of cell viability of BV-2 microglia cells, but it was not significant statistically. The group treated with 0.5 mg/ml of Puerariae Radix revealed significant increase of cell viability of BV-2 microglia cells damaged by nitric oxide. The groups treated with 5 and 50 mg/ml of Puerariae Radix demonstrated increases of cell viabilities of BV-2 microglia cells, but these were not significant statistically. These results suggested that Puerariae Radix revealed neuroprotective effects through the control effect of apoptosis and oxidative damages.