• ETRI Journal
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• v.44 no.3
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• pp.361-378
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• 2022

In 5G, the required target for interruption time during a handover (HO) is 0 ms. However, when a handover failure (HOF) occurs, the interruption time increases significantly to more than hundreds of milliseconds. Therefore, to fulfill the requirement in as many scenarios as possible, we need to minimize HOF rate as close to zero as possible. 3GPP has recently introduced conditional HO (CHO) to improve mobility robustness. In this study, we propose "ZEro handover failure with Unforced and automatic time-to-execute Scaling" (ZEUS) algorithm to optimize HO parameters easily in the CHO. Analysis and simulation results demonstrate that ZEUS can achieve a zero HOF rate without increasing the ping-pong rate. These two metrics are typically used to assess an HO algorithm because there is a tradeoff between them. With the introduction of the CHO, which solves the tradeoff, only these two metrics are insufficient anymore. Therefore, to evaluate the optimality of an HO algorithm, we define a new integrated HO performance metric, mobility-aware average effective spectral efficiency (MASE). The simulation results show that ZEUS provides higher MASE than LTE and other CHO variants.

• The Korean Journal of Nuclear Medicine
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• v.31 no.4
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• pp.433-439
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• 1997

$^{166}Ho$-chitosan complex, or $^{166}Ho$-CHICO, is a candidate pharmaceutical for intracavitary radiation therapy of cystic brain tumors because of the desirable nuclear characteristics of $^{166}Ho$ for therapeutic use and the suitable biological and chemical characteristics of chitosan, not to mention its ready producibility The amount of $^{166}Ho$-CHICO to be administered to obtain the goal therapeutic effect can be suggested by predicting the dose to the cyst wall for a varying pharmaceutical dose. When $^{166}Ho$-CHICO is infused into the cyst, the major part of the energy delivery by beta particles emitted from $^{166}Ho$ occurs in the cyst wall within 4mm in depth from the cyst wall surface. Also, realizing the attachment of $^{166}Ho$-CHICO to the cyst wall surface would change the predictions of dose to the cyst wall.

• Journal of the Korean Society of Food Science and Nutrition
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• v.41 no.9
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• pp.1226-1234
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• 2012

3,4,3',5'-Tetrahydroxy-trans-stilbene (piceatannol) is a derivative of resveratrol with a variety of biological activities, including anti-inflammatory, anti-proliferative, and anti-cancer activities. We assessed the mechanisms by which piceatannol inhibits inflammatory responses using lipopolysaccharide (LPS)-treated Raw264.7 murine macrophages. Piceatannol (0~10 ${\mu}mol/L$) decreased LPS-induced release of nitric oxide, tumor necrosis factor (TNF)-${\alpha}$, interleukin (IL)-6, IL-$1{\beta}$, and inhibited LPS-induced protein expression of inducible nitric oxide synthase (iNOS). Activation of nuclear factor-kappaB (NF-${\kappa}B$), activator protein (AP)-1, and signal transducer and activator of transcription 3 (STAT3) are crucial steps during an inflammatory response. Piceatannol prevented LPS-induced degradation of inhibitor of ${\kappa}B$ ($I{\kappa}B$), translocation of p65 to the nucleus, and phosphorylation of stress-activated protein kinase/c-Jun NH2-terminal kinase (SAPK/JNK). Additionally, piceatannol inhibited LPS-induced phosphorylation of STAT3 and IL-6-induced translocation of STAT3 to the nucleus. Furthermore, piceatannol increased the protein and mRNA levels of hemeoxygenase (HO)-1, the rate-limiting enzyme of heme catabolism that plays a critical role in mediating antioxidant and anti-inflammatory effects. Piceatannol further induced antioxidant response elements (ARE)-driven luciferase activity in Raw264.7 cells transfected with an ARE-luciferase reporter construct containing the enhancer 2 and minimal promoter region of HO-1. These results suggest that piceatannol exerts anti-inflammatory effects via the down-regulation of iNOS expression and up-regulation of HO-1 expression.

• Venture DIGEST
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• s.32
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• pp.3-3
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• 2003

• 열린충남
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• s.57
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• pp.6-10
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• 2012

• 건강소식
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• v.3 no.3 s.18
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• pp.18-20
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• 1975

• 환경사랑
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• s.46
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• pp.3-3
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• 2006

• KLA journal
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• v.51 no.3
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• pp.2-3
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• 2010

• Atmosphere
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• v.27 no.3
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• pp.375-375
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• 2017

• KLA journal
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• v.18 no.9
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• pp.3-3
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• 1977