References
- Bachmann IM, Puntervoll HE, Otte AP, Akslen LA (2008). Loss of BMI1 expression is associated with clinical progress of malignant melanoma. Modern Pathology, 21, 583-90. https://doi.org/10.1038/modpathol.2008.17
- Casas E, Kim J, Bendesky A, et al (2011). Snail2 is an essential mediator of TWIST1-induced epithelial mesenchymal transition and metastasis. Cancer Res, 71, 245-54.
- Gao XH, Yang XQ, Wang BC, et al (2013). Overexpression of twist and matrix metalloproteinase-9 with metastasis and prognosis in gastric cancer. Asian Pac J Cancer Prev, 14, 5055-60. https://doi.org/10.7314/APJCP.2013.14.9.5055
- Guo BH, Feng Y, Zhang R, et al (2011). BMI1 promotes invasion and metastasis, and its elevated expression is correlated with an advanced stage of breast cancer. Mol Cancer, 10, 10. https://doi.org/10.1186/1476-4598-10-10
- Guo W, Keckesova Z, Donaher JL, et al (2012). SNAI2 and Sox9 cooperatively determine the mammary stem cell state. Cell, 148, 1015-28. https://doi.org/10.1016/j.cell.2012.02.008
- Kouzmina M, Hayry V, Leikola J, et al (2012). BMI1 expression identifies subtypes of merkel cell carcinoma. Virchows Archiv, 461, 647-53 https://doi.org/10.1007/s00428-012-1327-7
- Majima Y, Hirakawa S, Kito Y, et al (2012). TWIST1 as a possible biomarker for metastatic basal cell carcinoma. Acta dermato-venereologica, 92, 585-63. https://doi.org/10.2340/00015555-1474
- Mihic Probst D, Kuster A, Kilgus S, et al (2007). Consistent expression of the stem cell renewal factor BMI1 in primary and metastatic melanoma. Int J Cancer, 121, 1764-70. https://doi.org/10.1002/ijc.22891
- Mikheeva SA, Mikheev AM, Petit A, et al (2010). TWIST1 promotes invasion through mesenchymal change in human glioblastoma. Mol Cancer, 9, 194. https://doi.org/10.1186/1476-4598-9-194
- Moad AI, Tan ML, Kaur G, Mabruk M (2012). Lack of Increased P15INK4B protein expression in basal cell carcinomas. Asian Pac J Cancer Prev, 13, 6239-44. https://doi.org/10.7314/APJCP.2012.13.12.6239
- Patel KJ, Rhodus N, Patel D, et al (2007). Role of SNAI2 in head and neck squamous cell carcinoma. Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology, and Endodontology, 103, 792-7.
- Reinisch CM, Uthman A, Erovic BM, Pammer J (2007). Expression of BMI1 in normal skin and inflammatory and neoplastic skin lesions. J Cutan Pathol, 34, 174-80. https://doi.org/10.1111/j.1600-0560.2006.00587.x
- Schwartz YB, Pirrotta V (2007). Polycomb silencing mechanisms and the management of genomic programmes. Nat Rev Genet, 8, 9-22.
- Shirley SH, Greene VR, Duncan LM, et al (2012). SNAI2 expression during melanoma progression. Am J Pathol, 180, 2479-89. https://doi.org/10.1016/j.ajpath.2012.02.014
- Vander Lugt NM, Domen J, Linders K, et al (1994). Posterior transformation, neurological abnormalities, and severe hematopoietic defects in mice with targeted deletion of BMI1 proto-oncogene. Genes Dev, 8, 757-69. https://doi.org/10.1101/gad.8.7.757
- Wang WS, Yu SL, Yang XS, et al (2013). Expression and significance of twist and E-cadherin in ovarian cancer tissues. Asian Pac J Cancer Prev, 14, 669-72. https://doi.org/10.7314/APJCP.2013.14.2.669
- Yang HW, Menon LG, Black PM, et al (2010). SNAI2/SLUG promotes growth and invasion in human gliomas. BMC Cancer, 10, 301. https://doi.org/10.1186/1471-2407-10-301
- Yang J, Mani SA, Donaher JL, et al (2004). Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis. Cell, 117, 927-96. https://doi.org/10.1016/j.cell.2004.06.006
- Yang MH, Hsu DS, Wang HW, et al (2010). BMI1 is essential in TWIST1-induced epithelial-mesenchymal transition. Nat Cell Biol, 10, 982-92.
- Zhu QC, Gao RY, Wu W, Qin HL (2013). Epithelialmesenchymal transition and its role in the pathogenesis of colorectal cancer. Asian Pac J Cancer Prev, 14, 2689-98. https://doi.org/10.7314/APJCP.2013.14.5.2689
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