References
- Bauer, J., Huitinga, I., Zhao, W., Lassmann, H., Hickey, W. F. and Dijkstra, C. D. The role of macrophages, perivascular cells, and microglial cells in the pathogenesis of experimental autoimmune encephalomyelitis. Glia. 1995, 15, 437-446
- Cooper, D. N. Galectinomics: finding themes in complexity. Biochim. Biophys. Acta. 2002, 1572, 209-231
- Demetriou, M., Granovsky, M., Quaggin, S. and Dennis, J. W. Negative regulation of T-cell activation and autoimmunity by Mgat5 N-glycosylation. Nature. 2001, 409, 733-739
- Lee, Y. K., Kang, H. E. and Woo, H. J. Expression of galectin-3 in rat brain. Korean J. Vet. Res. 2004, 44, 83-88
- Maeda, N., Kawada, N., Seki, S., Arakawa, T., Ikeda, K., Iwao, H., Okuyama, H., Hirabayashi, J., Kasai, K. and Yoshizato, K. Stimulation of proliferation of rat hepatic stellate cells by galectin-1 and galectin-3 through different intracellular signaling pathways. J. Biol. Chem. 2003, 278, 18938-18944
- Moon, B. K., Lee, Y. J., Battle, P., Jessup, J. M., Raz, A. and Kim, H. R. Galectin-3 protects human breast carcinoma cells against nitric oxide-induced apoptosis; implication of galectin-3 function during metastasis. Am. J. Pathol. 2001, 159, 1055-1060
- Nguyen, K. B., McCombe, P. A. and Pender, M. P. Increased apoptosis of T lymphocyte and macrophages in the central and peripheral nervous systems of Lewis rats with exprimental autoimmune encephalomyelitis treated with dexamethasone. J. Neuropathol. Exp. Neurol. 1997, 56, 58-69
- Raine, C. S. and Traugott, U. Experimental autoimmune demyelination. Chronic relapsing models and their therapeutic implications for multiple sclerosis. Ann. N. Y. Acad. Sci. 1984, 436, 33-51
- Regan, L. J., Dodd, J., Barondes, S. H. and Jessell, T. M. Selective expression of endogenous lactosebinding lectins and lactoseries glycoconjugates in subsets of rat sensory neurons. Proc. Natl. Acad. Sci. U.S.A. 1986, 83, 2248-2252
- Reichert, F. and Rotshenker, S. Galectin-3/MAC-2 in experimental allergic encephalomyelitis. Exp. Neurol. 1999, 160, 508-514
- Schmied, M., Breitschopf, H., Gold, R., Zischler, H., Rothe, G., Wekerle, H. and Lassmann, H. Apoptosis of T lymphocytes in experimental autoimmune encephalomyelitis. Evidence for programmed cell death as a mechanism to control inflammation in the brain. Am. J. Pathol. 1993, 143, 446-452
- Sedgwick, J. D. and Mason, D. W. The mechanism of inhibition of experimental allergic encephalomyelitis in the rat by monoclonal antibody against CD4. J.Neuroimmunol. 1986, 13, 217-232
- Sekiguchi, Y., Ichikawa, M., Inoue, A., Itoh, M. and Koh, C. S. Brain-derived gangliosides suppress the chronic relapsing-remitting experimental autoimmuneencephalomyelitis in NOD induced with myelin oligodendrocyte glycoprotein peptide. J. Neuroimmunol. 2001, 116, 196-205
- Shin, T., Kojima, T., Tanuma, N., Ishihara, Y. and Matsumoto, Y. The subarachnoid space as a site for precursor T cell proliferation and effector T cell selection in experimental autoimmune encephalomyelitis. J. Neuroimmunol. 1995, 56, 171-178
- Song, Y. K., Billiar, T. R. and Lee, Y. J. Role of galectin-3 in breast cancer metastasis: involvement of nitric oxide. Am. J. Pathol. 2002, 160, 1069-1075
- Tanuma, N., Kojima, T., Shin, T., Aikawa, Y., Kohji, T., Ishihara, Y. and Matsumoto, Y. Competitive PCR quantification of pro- and anti-inflammatory cytokine mRNA in the central nervous system during autoimmune encephalomyelitis. J. Neuroimmunol. 1997, 73, 197-206
- Woo, H. J., Shaw, L. W., Messier, J. M. and Mercurio, A. M. The major non-integrin laminin binding protein of macrophases is identical to carbohydrate binding protein 35 (Mac-2). J. Biol. Chem. 1990, 265, 7097-7099
- Yang, R. Y., Hsu, D. K. and Liu, F. T. Expression of galectin-3 modulates T-cell growth and apoptosis. Proc. Natl. Acad. Sci. U.S.A. 1996, 93, 6737-6742