Detection of Single Nucleotide Polymorphism in Human IL-4 Receptor by PCR Amplification of Specific Alleles

  • Hwang, Sue Yun (Institute of Immunobiology, Catholic Institutes of Medical Science, The Catholic University of Korea Medical College) ;
  • Kim, Seung Hoon (Institute of Immunobiology, Catholic Institutes of Medical Science, The Catholic University of Korea Medical College) ;
  • Hwang, Sung Hee (Institute of Immunobiology, Catholic Institutes of Medical Science, The Catholic University of Korea Medical College) ;
  • Cho, Chul Soo (Center for Rheumatic Disease, Kangnam St. Mary's Hospital, The Catholic University of Korea Medical College) ;
  • Kim, Ho Youn (Center for Rheumatic Disease, Kangnam St. Mary's Hospital, The Catholic University of Korea Medical College)
  • Published : 2001.06.01

Abstract

A key aspect of genomic research in the “post-genome era”is to associate sequence variations with heritable phenotypes. The most common variations in the human genome are single nucleotide polymorphisms (SNPs) that occur approximately once in every 500 to 1,000 bases. Although analyzing the phenotypic outcome of these SNPs is crucial to facilitate large-scale association studies of genetic diseases, detection of SNPs from an extended number of human DNA samples is often difficult, labor-intensive and time-consuming. Recent development in SNP detection methods using DNA microarrays and mass spectrophotometry has allowed automated high throughput analyses, but such equipments are not accessible to many scientists. In this study, we demonstrate that a simple PCR-based method using primers with a mismatched base at the 3'-end provides a fast and easy tool to identify known SNPs from human genomic DNA in a regular molecular biology laboratory. Results from this PCR amplification of specific alleles (PASA) analysis efficiently and accurately typed the Q576R polymorphism of human IL4 receptor from the genomic DNAs of 29 Koreans, including 9 samples whose genotype could not be discerned by the conventiona1 PCR-SSCP (single strand conformation polymorphism) method. Given the increasing attention to disease-associated polymorphisms in genomic research, this alternative technique will be very useful to identify SNPs in large-scale population studies.

Keywords

References

  1. Collins FS, Patrinos A, Jordan E, Chakravarti A, Gesteland R, and Walters L (1998) new goals for the U.S Human Genome Project: 1998-2003. Science 282: 682-689 https://doi.org/10.1126/science.282.5389.682
  2. Cooper DN, Smith BA, Cooke H, Niemann S an Schdictke J (1985) an estimate of unique sequence heterozygosity in the human genome. Hum Genet 69: 201-205 https://doi.org/10.1007/BF00293024
  3. Harding RM, Fullerton SM, Griffith RC, Bond J, Cox MJ, Schneider JA, Moulin DS and Clegg JB (1997) Archaic African and Asian lineage in the genetic ancestry of modern humans. Am J Hum Genet 60: 772-789
  4. Hershey G, Friedrich M, Esswein L, Thomas M and Chatila T (1997) The association of atopy with a gain-of-function mutator in the a subunit of the interleukin 4 receptor. N Engl J Med 337: 1720-1725 https://doi.org/10.1056/NEJM199712113372403
  5. Hwang SH, Youn J, Cho CS, Min JK, Kim WU, Park SH and Kim HY (2000) Assocoation of the interleukin-4 receptor a variant Q576R with Th1/Th2 imbalance in connective tissue disease. Immunogenetics 51: 746-746
  6. Kruglyak L (1997) The use of a genetic map of biallelic markers in linkage studies. Nat Genet 17: 21-24 https://doi.org/10.1038/ng0997-21
  7. Sommer SS, Cassady JD, Sobell JL, and Bottema CD (1989) A Novel method for detecting point mutations or polymorphisms and its application to population screen for carriers of phenylketonuria. Mayo Clin Proc 64: 1361-1372
  8. Zetterquist H and Olerup O (1995) A novel DRB1 allele (HLA-DRB1*1318) featuring a DR8 associated sequence motif on an FR52 haplotype. Tissue Antigens 46: 337-339