The Korean Journal of Pesticide Science (농약과학회지)
- Volume 2 Issue 3
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- Pages.1-20
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- 1998
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- 1226-6183(pISSN)
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- 2287-2051(eISSN)
Herbicidal action mechanism of chlorsulfuron
Acetolactate synthase 저해 제초제인 chlorsulfuron의 작용기작
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Kim, Song-Mun
(Institute for Agricultural Science Research, College of Agriculture and Life Science, Kangwon National University) ;
- Kim, Yong-Ho (Division of Biological Environment, College of Agriculture and Life Science, Kangwon National University) ;
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Hur, Jang-Hyun
(Division of Biological Environment, College of Agriculture and Life Science, Kangwon National University) ;
- Han, Dae-Sung (Division of Biological Environment, College of Agriculture and Life Science, Kangwon National University)
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김성문
(강원대학교 농업생명과학대학 농업과학연구소) ;
- 김용호 (강원대학교 농업생명과학대학 자원생물환경학부) ;
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허장현
(강원대학교 농업생명과학대학 자원생물환경학부) ;
- 한대성 (강원대학교 농업생명과학대학 자원생물환경학부)
- Published : 1998.12.30
Abstract
Chlorsulfuron, one of sulfonylurea herbicides acts through inhibition of acetolactate syuthase (EC 4.1.3.18; ALS, also known as acetohydroxyacid synthase) in the branched-chain amino acid biosynthesis process. After chlorsulfuron-ALS interaction, many physiological and metabolic disruptions occur in plants. However, it is not clear how this chlorsulfuron-ALS interaction affects those physiological and metabolic processes and how this interaction leads subsequently to plant death. Several researchers suggested that the death of chlorsulfuron-treated plants might be due to a shortage of the branched-chain amino acids, an accumulation of toxic metabolites, and/or a depletion of photoassimilates. It remains as a mystery presently, however, if such changes result in the plant death. In this review, we discussed how the chlorsulfuran-ALS interaction leads to physiological and metabolic disruptions in plants.