Study on Syndrome Differentiation of Gastritis by Korean Standard Classification of Dsease and Cause of Death

한국표준질병 사인분류에 따른 위염(胃炎)의 한의학적 변증 연구

  • Park, Mi Sun (Department of Korean Pathology, College of Korean Medicine, Wonkwang University) ;
  • Kim, Yeong Mok (Department of Korean Pathology, College of Korean Medicine, Wonkwang University)
  • 박미선 (원광대학교 한의과대학 한방병리학교실) ;
  • 김영목 (원광대학교 한의과대학 한방병리학교실)
  • Received : 2017.04.13
  • Accepted : 2017.10.10
  • Published : 2017.10.25


This article is for understanding relations between the classifications of gastritis and syndrome differentiation types of Korean Medicine through research on syndrome differentiations of clinically applied gastritis and literature of Korean Medicine. Clinical papers were searched in China Academic Journals(CAJ) of China National Knowledge Infrastructure(CNKI) from 1995 to 2015. Conclusions are as follows. First, disease mechanism of chronic gastritis are qi stagnation, damp stagnation, heat obstruction, blood stasis obstruction, yin damage, damage to collaterals with healthy qi deficiency and pathogenic qi. And qi movement stagnation is shown through the status of chronic gastritis. Second, chronic superficial gastritis belongs to qi aspect syndrome and mainly pathogen excess syndrome. And the key mechanisms are congestion and disharmony of stomach qi sometimes combined with liver depression, food accumulation and dampness-heat. Third, chronic atrophic gastritis belongs to qi-blood syndrome and deficiency-excess complex syndrome with the root of spleen qi deficiency and stomach yin deficiency and the tip of blood stasis, qi stagnation. And key mechanism is damage to collaterals with healthy qi deficiency and toxin-blood stasis. Forth, pathogen excess syndromes are shown at the early stage of chronic gastritis and healthy qi deficiency syndromes after the middle stage. Qi deficiency is shown at the beginning of the disease and yin deficiency at the late stage. And qi deficiency is related with superficial gastritis and yin deficiency with atrophic gastritis.


Supported by : 한국연구재단


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